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@ARTICLE{Strelow:1008541,
author = {Strelow, Joshua N. and Dembek, Till A. and Baldermann, Juan
C. and Andrade, Pablo and Fink, Gereon R. and
Visser-Vandewalle, Veerle and Barbe, Michael T.},
title = {{L}ow beta-band suppression as a tool for {DBS} contact
selection for akinetic-rigid symptoms in {P}arkinson's
disease},
journal = {Parkinsonism $\&$ related disorders},
volume = {112},
issn = {1353-8020},
address = {Amsterdam [u.a.]},
publisher = {Elsevier Science},
reportid = {FZJ-2023-02378},
pages = {105478 -},
year = {2023},
abstract = {AbstractBackground: Suppression of pathologically altered
activity in the beta-band has previously been suggested as a
biomarker for feedback-based neurostimulation in subthalamic
deep brain stimulation (STN-DBS) for Parkinson's Disease
(PD).Objective: To assess the utility of beta-band
suppression as a tool for contact selection in STN-DBS for
PD.Methods: A sample of seven PD patients (13 hemispheres)
with newly implanted directional DBS leads of the STN were
recorded during a standardized monopolar contact review
(MPR). Recordings were received from contact pairs adjacent
to the stimulation contact. The degree of beta-band
suppression for each investigated contact was then
correlated to the respective clinical results. Additionally,
we have implemented a cumulative ROC analysis, to test the
predictive value of beta-band suppression on the clinical
efficacy of the respective contacts.Results: Stimulation
ramping led to frequency-specific changes in the beta-band,
while lower frequencies remained unaffected. Most
importantly, our results showed that the degree of low
beta-band suppression from baseline activity (stimulation
off) served as a predictor for clinical efficacy of the
respective stimulation contact. In contrast suppression of
high beta-band activity yielded no predictive
power.Conclusion: The degree of low beta-band suppression
can serve as a time-saving, objective tool for contact
selection in STN-DBS.Keywords: DBS programming; Deep brain
stimulation (DBS); Local field potential (LFP); Nucleus
subthalamicus (STN).},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {5251 - Multilevel Brain Organization and Variability
(POF4-525) / DFG project 431549029 - SFB 1451:
Schlüsselmechanismen normaler und krankheitsbedingt
gestörter motorischer Kontrolle (431549029)},
pid = {G:(DE-HGF)POF4-5251 / G:(GEPRIS)431549029},
typ = {PUB:(DE-HGF)16},
pubmed = {37331065},
UT = {WOS:001025640500001},
doi = {10.1016/j.parkreldis.2023.105478},
url = {https://juser.fz-juelich.de/record/1008541},
}