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@ARTICLE{Reimann:1009298,
author = {Reimann, Gerion M. and Küppers, Vincent and Camilleri,
Julia and Hoffstaedter, Felix and Langner, Robert and Laird,
Angela R. and Fox, Peter T. and Spiegelhalder, Kai and
Eickhoff, Simon B. and Tahmasian, Masoud},
title = {{C}onvergent abnormality in the subgenual anterior
cingulate cortex in insomnia disorder: {A} revisited
neuroimaging meta-analysis of 39 studies},
journal = {Sleep medicine reviews},
volume = {71},
issn = {1087-0792},
address = {Kidlington, Oxford [u.a.]},
publisher = {Elsevier},
reportid = {FZJ-2023-02748},
pages = {101821 -},
year = {2023},
abstract = {The neurobiological underpinnings of insomnia disorder (ID)
are still poorly understood. A previous meta-analysis
conducted by our research group in 2018 revealed no
consistent regional alterations based on the limited number
of eligible studies. Given the number of studies published
during the last few years, we revisited the meta-analysis to
provide an update to the field. Following the best-practice
guidelines for conducting neuroimaging meta-analyses, we
searched several databases (PubMed, Web of Science, and
BrainMap) and identified 39 eligible structural and
functional studies, reporting coordinates reflecting
significant group differences between ID patients and
healthy controls. A significant convergent regional
alteration in the subgenual anterior cingulate cortex
(sgACC) was observed using the activation likelihood
estimation algorithm. Behavioural decoding using the
BrainMap database indicated that this region is involved in
fear-related emotional and cognitive processing. The sgACC
showed robust task-based co-activation in meta-analytic
connectivity modelling and task-free functional connectivity
in a resting-state functional connectivity analysis with the
main hubs of the salience and default mode networks,
including the posterior cingulate cortex and dorsal ACC,
amygdala, hippocampus, and medial prefrontal cortex.
Collectively, the findings from this large-scale
meta-analysis suggest a critical role of the sgACC in the
pathophysiology of ID.},
cin = {INM-7},
ddc = {780},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {37481961},
UT = {WOS:001049533700001},
doi = {10.1016/j.smrv.2023.101821},
url = {https://juser.fz-juelich.de/record/1009298},
}