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@INPROCEEDINGS{Shimoura:1009485,
author = {Shimoura, Renan and Roque, Antonio Carlos and van Albada,
Sacha},
title = {{V}isual alpha generators in a full-density spiking
thalamocortical model},
reportid = {FZJ-2023-02822},
year = {2023},
note = {References:[1] Clayton, M. S., Yeung, N., $\&$ Cohen
Kadosh, R. (2017). European Journal of Neuroscience, 48(7),
2498-2508.[2] Silva, L., Amitai, Y., $\&$ Connors, B.
(1991). Science, 251(4992), 432–435.[3] Roberts, J. A.,
$\&$ Robinson, P. A. (2008). Journal of Theoretical Biology,
253(1), 189–201.[4] Van Kerkoerle, T., Self, M. W.,
Dagnino, B., Gariel-Mathis, M. A., Poort, J., Van Der Togt,
C., $\&$ Roelfsema, P. R. (2014). Proceedings of the
National Academy of Sciences, 111(40), 14332-14341.[5]
Bollimunta, A., Mo, J., Schroeder, C. E., $\&$ Ding, M.
(2011). Journal of Neuroscience, 31(13), 4935-4943.},
abstract = {The alpha rhythm (~10 Hz) is one of the most prominent
features in waking electroencephalograms of a variety of
mammals. It is mainly observed in occipitoparietal regions
during the eyes-closed resting state. Although alpha is
strongly associated with reduced visual attention, it is
also related to other functions such as regulation of timing
and temporal resolution of perception, and transmission
facilitation of predictions to visual cortex [1].
Understanding how and where this rhythm is generated can
elucidate its functions. Even today there is no definitive
answer to this question, though several hypotheses put
forward thalamus and cortex as possible protagonists.In this
work, we built a full-density spiking thalamocortical model,
including the primary visual cortex (V1) and the lateral
geniculate nucleus (LGN), to study two potential alpha
rhythm generators: 1) rhythmic bursts produced by pyramidal
neurons in L5 at around 10 Hz [2]; 2) a thalamocortical loop
delay of approximately 100 ms, as suggested in mean-field
models [3]. The cortical component of our model covers 1 mm2
of V1 surface and is divided into four layers (L2/3, L4, L5,
and L6), each containing excitatory and inhibitory
populations. The thalamic network comprises an excitatory
and an inhibitory population. All neurons were simulated by
the adaptive exponential integrate-and-fire model. Cortical
neurons in L4 and L6 receive thalamocortical connections,
and L6 neurons provide feedback projections to the thalamus.
We performed all network simulations using the NEST
simulator. The resulting spiking activity was recorded and
compared with experimental data by means of power spectra
and Granger Causality (GC) analysis.Our results show that
both mechanisms are capable of generating and spreading
alpha oscillations through the layers, but with different
laminar patterns. In Hypothesis 1, the GC analysis suggests
that the alpha rhythm mainly originates in L5 and L2/3, as
reported in experimental studies with macaques where
top-down feedback alpha was observed [4]. On the other hand,
Hypothesis 2 points to L4 and L6 as the primary source
layers, which may be interpreted as feedforward alpha
propagation and matches laminar patterns observed in another
macaque study [5]. Furthermore, combining both mechanisms
resulted in a summation of effects, with GC in the alpha
range emanating from all layers. Thus, our findings suggest
that the two mechanisms may contribute differently to alpha
rhythms, with distinct laminar patterns, and may be
expressed either separately or in tandem under different
conditions.},
month = {Jul},
date = {2023-07-15},
organization = {32nd Annual Computational Neuroscience
Meeting CNS*2023, Leipzig (Germany), 15
Jul 2023 - 19 Jul 2023},
subtyp = {Other},
cin = {INM-6 / IAS-6 / INM-10},
cid = {I:(DE-Juel1)INM-6-20090406 / I:(DE-Juel1)IAS-6-20130828 /
I:(DE-Juel1)INM-10-20170113},
pnm = {5231 - Neuroscientific Foundations (POF4-523) / HBP SGA2 -
Human Brain Project Specific Grant Agreement 2 (785907) /
HBP SGA3 - Human Brain Project Specific Grant Agreement 3
(945539) / DFG project 347572269 - Heterogenität von
Zytoarchitektur, Chemoarchitektur und Konnektivität in
einem großskaligen Computermodell der menschlichen
Großhirnrinde (347572269)},
pid = {G:(DE-HGF)POF4-5231 / G:(EU-Grant)785907 /
G:(EU-Grant)945539 / G:(GEPRIS)347572269},
typ = {PUB:(DE-HGF)24},
doi = {10.34734/FZJ-2023-02822},
url = {https://juser.fz-juelich.de/record/1009485},
}