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@ARTICLE{HerreraRivero:1010147,
author = {Herrera-Rivero, Marisol and Gutiérrez-Fragoso, Karina and
Thalamuthu, Anbupalam and Amare, Azmeraw T. and Adli, Mazda
and Akiyama, Kazufumi and Akula, Nirmala and Ardau,
Raffaella and Arias, Bárbara and Aubry, Jean-Michel and
Backlund, Lena and Bellivier, Frank and Benabarre, Antonio
and Bengesser, Susanne and Abesh, Bhattacharjee and
Biernacka, Joanna and Birner, Armin and Cearns, Micah and
Cervantes, Pablo and Chen, Hsi-Chung and Chillotti, Caterina
and Cichon, Sven and Clark, Scott and Colom, Francesc and
Cruceanu, Cristiana and Czerski, Piotr and Dalkner, Nina and
Degenhardt, Franziska and Zompo, Maria Del and DePaulo, J.
Raymond and Etain, Bruno and Falkai, Peter and
Ferensztajn-Rochowiak, Ewa and Forstner, Andreas J. and
Frank, Josef and Frisen, Louise and Frye, Mark and
Fullerton, Janice and Gallo, Carla and Gard, Sebastien and
Garnham, Julie and Goes, Fernando and Grigoroiu-Serbanescu,
Maria and Grof, Paul and Hashimoto, Ryota and Hasler, Roland
and Hauser, Joanna and Heilbronner, Urs and Herms, Stefan
and Hoffmann, Per and Hou, Liping and Hsu, YiHsiang and
Jamain, Stéphane and Jiménez, Esther and Kahn, Jean-Pierre
and Kassem, Layla and Kato, Tadafumi and Kelsoe, John and
Kittel-Schneider, Sarah and kuo, Po-Hsiu and Kurtz, Joachim
and Kusumi, Ichiro and König, Barbara and Laje, Gonzalo and
Landén, Mikael and Lavebratt, Catharina and Leboyer, Marion
and Leckband, Susan and Maj, Mario and Manchia, Mirko and
Marie-Claire, Cynthia and Martinsson, Lina and McCarthy,
Michael and McElroy, Susan L. and Millischer, Vincent and
Mitjans, Marina and Mondimore, Francis and Monteleone,
Palmiero and Nievergelt, Caroline and Novak, Tomas and
Nöthen, Markus and odonovan, claire and Ozaki, Norio and
Papiol, Sergi and Pfennig, Andrea and Pisanu, Claudia and
Potash, James and Reif, Andreas and Reininghaus, Eva and
Richard-Lepouriel, Hélène and Roberts, Gloria and Rouleau,
Guy and Rybakowski, Janusz K. and Schalling, Martin and
Schofield, Peter and Schubert, Klaus Oliver and Schulte, Eva
and SCHWEIZER, BARBARA and Severino, Giovanni and Shekhtman,
Tatyana and Shilling, Paul and Shimoda, Kazutaka and
Simhandl, Christian and slaney, claire and Squassina,
Alessio and Stamm, Thomas and Stopkova, Pavla and Streit,
Fabian and Ayele, Fasil and Tortorella, Alfonso and Turecki,
Gustavo and Veeh, Julia and Vieta, Eduard and Viswanath,
Biju and Witt, Stephanie and Zandi, Peter and Alda, Martin
and Bauer, Michael and McMahon, Francis and Mitchell, Philip
and Rietschel, Marcella and Schulze, Thomas and Baune,
Bernhard},
title = {{I}mmunogenetics of lithium response and psychiatric
phenotypes in patients with bipolar disorder},
reportid = {FZJ-2023-02975},
year = {2023},
abstract = {The link between bipolar disorder (BP) and immune
dysfunction remains controversial. While epidemiological
studies have long suggested an association, recent research
has found only limited evidence of such a relationship. To
clarify this, we investigated the contributions of
immune-relevant genetic factors to the response to lithium
(Li) treatment and the clinical presentation of BP. First,
we assessed the association of a large collection of
immune-related genes (4,925) with Li response, defined by
the Retrospective Assessment of the Lithium Response
Phenotype Scale (Alda scale), and clinical characteristics
in patients with BP from the International Consortium on
Lithium Genetics (ConLi + Gen, N = 2,374). Second, we
calculated here previously published polygenic scores (PGSs)
for immune-related traits and evaluated their associations
with Li response and clinical features. We found several
genes associated with Li response at p < 1x10 - 4 values,
including HAS3 , CNTNAP5 and NFIB . Network and functional
enrichment analyses uncovered an overrepresentation of
pathways involved in cell adhesion and intercellular
communication, which appear to converge on the well-known
Li-induced inhibition of GSK-3β. We also found various
genes associated with BP's age-at-onset, number of mood
episodes, and presence of psychosis, substance abuse and/or
suicidal ideation at the exploratory threshold. These
included RTN4 , XKR4 , NRXN1 , NRG1/3 and GRK5 .
Additionally, PGS analyses suggested serum FAS, ECP, TRANCE
and cytokine ligands, amongst others, might represent
potential circulating biomarkers of Li response and clinical
presentation. Taken together, our results support the notion
of a relatively weak association between immunity and
clinically relevant features of BP at the genetic level.},
cin = {INM-1},
cid = {I:(DE-Juel1)INM-1-20090406},
pnm = {5251 - Multilevel Brain Organization and Variability
(POF4-525)},
pid = {G:(DE-HGF)POF4-5251},
typ = {PUB:(DE-HGF)25},
doi = {10.21203/rs.3.rs-3068352/v1},
url = {https://juser.fz-juelich.de/record/1010147},
}