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@ARTICLE{Kriehuber:1010519,
author = {Kriehuber, Ralf and Simkó, Myrtill},
title = {{A}poptosis {I}nduction and {M}icronucleus {F}ormation
after {E}xposure to the {A}uger {E}lectron {E}mitter
{Z}inc-65 in a {H}uman {C}ell {L}ine},
journal = {Acta oncologica},
volume = {39},
number = {6},
issn = {0284-186X},
address = {Abingdon},
publisher = {Taylor $\&$ Francis Group},
reportid = {FZJ-2023-03102},
pages = {699 - 706},
year = {2000},
abstract = {Induction of apoptosis and micronucleus formation has been
studied in a transformed human squamous cell carcinoma cell
line (SCL-II) after exposure to the Auger electron emitter
Zinc-65 (65Zn) and after external low-LET radiation.
Exposure to non-radioactive Zn and unirradiated cells served
as controls. Studies on the cellular uptake of 65Zn2+ have
been carried out in vitro and conventional dosimetric models
have been applied to derive the absorbed radiation dose.
Auger electrons, generated during decay of 65Zn2+, are
strong inducers of micronuclei as well as of apoptosis, in
comparison to external low-LET irradiation. The relative
biological effectiveness has been determined and was found
to be in the range of 1.2-2.7 for the two investigated
biological endpoints, depending on which mathematical model
for describing the dose-effect curves was used. A
non-uniform distribution of intracellular Zn2 + was
observed, showing a strong signal in the perinuclear region.
We conclude that separate radiation weighting factors for
Auger electrons should be established depending on the
nuclide and its ability to interact with the DNA (e.g. 65Zn
by Zinc-finger proteins)},
cin = {S-US},
ddc = {610},
cid = {I:(DE-Juel1)S-US-20090406},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {11130007},
UT = {WOS:000165549800008},
doi = {10.1080/028418600750063758},
url = {https://juser.fz-juelich.de/record/1010519},
}