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@ARTICLE{SternkeHoffmann:1016953,
author = {Sternke-Hoffmann, Rebecca and Pauly, Thomas and Norrild,
Rasmus K. and Hansen, Jan and Tucholski, Florian and Høie,
Magnus Haraldson and Marcatili, Paolo and Dupré, Mathieu
and Duchateau, Magalie and Rey, Martial and Malosse,
Christian and Metzger, Sabine and Boquoi, Amelie and
Platten, Florian and Egelhaaf, Stefan U. and Chamot-Rooke,
Julia and Fenk, Roland and Nagel-Steger, Luitgard and Haas,
Rainer and Buell, Alexander K.},
title = {{W}idespread amyloidogenicity potential of multiple myeloma
patient-derived immunoglobulin light chains},
journal = {BMC biology},
volume = {21},
number = {1},
issn = {1741-7007},
address = {Heidelberg},
publisher = {Springer},
reportid = {FZJ-2023-03861},
pages = {21},
year = {2023},
abstract = {Background:In a range of human disorders such as multiple
myeloma (MM), immunoglobulin light chains (IgLCs) can be
produced at very high concentrations. This can lead to
pathological aggregation and deposition of IgLCs in
different tissues, which in turn leads to severe and
potentially fatal organ damage. However, IgLCs can also be
highly soluble and non-toxic. It is generally thought that
the cause for this differential solubility behaviour is
solely found within the IgLC amino acid sequences, and a
variety of individual sequence-related biophysical
properties (e.g. thermal stability, dimerisation) have been
proposed in different studies as major determinants of the
aggregation in vivo. Here, we investigate biophysical
properties underlying IgLC amyloidogenicity.Results:We
introduce a novel and systematic workflow, Thermodynamic and
Aggregation Fingerprinting (ThAgg-Fip), for detailed
biophysical characterisation, and apply it to nine different
MM patient-derived IgLCs. Our set of pathogenic IgLCs spans
the entire range of values in those parameters previously
proposed to define in vivo amyloidogenicity; however, none
actually forms amyloid in patients. Even more surprisingly,
we were able to show that all our IgLCs are able to form
amyloid fibrils readily in vitro under the influence of
proteolytic cleavage by co-purified
cathepsins.Conclusions:We show that (I) in vivo aggregation
behaviour is unlikely to be mechanistically linked to any
single biophysical or biochemical parameter and (II)
amyloidogenic potential is widespread in IgLC sequences and
is not confined to those sequences that form amyloid fibrils
in patients. Our findings suggest that protein sequence,
environmental conditions and presence and action of
proteases all determine the ability of light chains to form
amyloid fibrils in patients.},
cin = {IBI-7 / IBI-4},
ddc = {610},
cid = {I:(DE-Juel1)IBI-7-20200312 / I:(DE-Juel1)IBI-4-20200312},
pnm = {5241 - Molecular Information Processing in Cellular Systems
(POF4-524)},
pid = {G:(DE-HGF)POF4-5241},
typ = {PUB:(DE-HGF)36 / PUB:(DE-HGF)16},
pubmed = {36737754},
UT = {WOS:000940546800003},
doi = {10.1186/s12915-022-01506-w},
url = {https://juser.fz-juelich.de/record/1016953},
}