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@ARTICLE{Seger:1018436,
author = {Seger, Aline and Ophey, Anja and Doppler, Christopher E. J.
and Kickartz, Johanna and Lindner, Marie-Sophie and
Hommelsen, Maximilian and Fink, Gereon R. and Sommerauer,
Michael},
title = {{C}linical subtypes in patients with isolated {REM} sleep
behaviour disorder},
journal = {npj Parkinson's Disease},
volume = {9},
number = {1},
issn = {2373-8057},
address = {London [u.a.]},
publisher = {Nature Publ. Group},
reportid = {FZJ-2023-04811},
pages = {155},
year = {2023},
abstract = {Patients with Parkinson's disease (PD) show a broad
heterogeneity in dinical presentation, and subtypes may
already arise inprodromal disease stages. lsolated REM sleep
behaviour disorder (iRBD) is the most specific marker of
prodromal PD, but data ondinical subtyping of patients with
iRBD remain scarce. Therefore, this study aimed to identify
iRBD subtypes. We conductedcomprehensive dinical assessments
in 66 patients with polysomnography-proven iRBD, induding
motor and non-motorevaluations, and applied a two-step
duster analysis. Besides, we compared iRBD dusters to
matched healthy controls and relatedthe resulting duster
solution to cortical and subcortical grey matter volumes by
voxel-based morphometry analysis. We identifiedtwo distinct
subtypes of patients based on olfactory function, dominant
electroencephalography frequency, amount of REM sleepwithout
atonia, depressive symptoms, disease duration, and motor
functions. One iRBD duster (Cluster 1, late
onset-aggressive)was characterised by higher non-motor
symptom burden despite shorter disease duration than the
more benign subtype (Cluster II,early onset-benign). Motor
functions were comparable between the dusters. Patients from
Cluster I were significantly older atiRBD onset and
exhibited a widespread reduction of cortical grey matter
volume compared to patients from Cluster II. In
condusion,our findings suggest the existence of dinical
subtypes already in the prodromal stage of PD. Future
longitudinal studies arewarranted that replicate these
findings and investigate the risk of the more aggressive
phenotype for earlier phenoconversion anddementia
development.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525) / DFG
project 431549029 - SFB 1451: Schlüsselmechanismen normaler
und krankheitsbedingt gestörter motorischer Kontrolle
(431549029) / DFG project 491111487 -
Open-Access-Publikationskosten / 2022 - 2024 /
Forschungszentrum Jülich (OAPKFZJ) (491111487)},
pid = {G:(DE-HGF)POF4-5252 / G:(GEPRIS)431549029 /
G:(GEPRIS)491111487},
typ = {PUB:(DE-HGF)16},
pubmed = {37978183},
UT = {WOS:001104547000001},
doi = {10.1038/s41531-023-00598-7},
url = {https://juser.fz-juelich.de/record/1018436},
}