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@ARTICLE{Passiatore:1019091,
author = {Passiatore, Roberta and Antonucci, Linda A. and DeRamus,
Thomas P. and Fazio, Leonardo and Stolfa, Giuseppe and
Sportelli, Leonardo and Kikidis, Gianluca C. and Blasi,
Giuseppe and Chen, Qiang and Dukart, Juergen and Goldman,
Aaron L. and Mattay, Venkata S. and Popolizio, Teresa and
Rampino, Antonio and Sambataro, Fabio and Selvaggi,
Pierluigi and Ulrich, William and Weinberger, Daniel R. and
Bertolino, Alessandro and Calhoun, Vince D. and Pergola,
Giulio},
title = {{C}hanges in patterns of age-related network connectivity
are associated with risk for schizophrenia},
journal = {Proceedings of the National Academy of Sciences of the
United States of America},
volume = {120},
number = {32},
issn = {0027-8424},
address = {Washington, DC},
publisher = {National Acad. of Sciences},
reportid = {FZJ-2023-05144},
pages = {e2221533120},
year = {2023},
abstract = {Alterations in fMRI-based brain functional network
connectivity (FNC) are associated with schizophrenia (SCZ)
and the genetic risk or subthreshold clinical symptoms
preceding the onset of SCZ, which often occurs in early
adulthood. Thus, age-sensitive FNC changes may be relevant
to SCZ risk-related FNC. We used independent component
analysis to estimate FNC from childhood to adulthood in
9,236 individuals. To capture individual brain features more
accurately than single-session fMRI, we studied an average
of three fMRI scans per individual. To identify potential
familial risk–related FNC changes, we compared age-related
FNC in first-degree relatives of SCZ patients mostly
including unaffected siblings (SIB) with neurotypical
controls (NC) at the same age stage. Then, we examined how
polygenic risk scores for SCZ influenced risk-related FNC
patterns. Finally, we investigated the same risk-related FNC
patterns in adult SCZ patients (oSCZ) and young individuals
with subclinical psychotic symptoms (PSY). Age-sensitive
risk-related FNC patterns emerge during adolescence and
early adulthood, but not before. Young SIB always followed
older NC patterns, with decreased FNC in a
cerebellar–occipitoparietal circuit and increased FNC in
two prefrontal–sensorimotor circuits when compared to
young NC. Two of these FNC alterations were also found in
oSCZ, with one exhibiting reversed pattern. All were linked
to polygenic risk for SCZ in unrelated individuals (R2
varied from 0.02 to 0.05). Young PSY showed FNC alterations
in the same direction as SIB when compared to NC. These
results suggest that age-related neurotypical FNC correlates
with genetic risk for SCZ and is detectable with MRI in
young participants.},
cin = {INM-7},
ddc = {500},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {37527347},
UT = {WOS:001083463000007},
doi = {10.1073/pnas.2221533120},
url = {https://juser.fz-juelich.de/record/1019091},
}