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@ARTICLE{Schinz:1019092,
author = {Schinz, David and Schmitz-Koep, Benita and Zimmermann,
Juliana and Brandes, Elin and Tahedl, Marlene and Menegaux,
Aurore and Dukart, Jürgen and Zimmer, Claus and Wolke,
Dieter and Daamen, Marcel and Boecker, Henning and Bartmann,
Peter and Sorg, Christian and Hedderich, Dennis M.},
title = {{I}ndirect evidence for altered dopaminergic
neurotransmission in very premature‐born adults},
journal = {Human brain mapping},
volume = {44},
number = {15},
issn = {1065-9471},
address = {New York, NY},
publisher = {Wiley-Liss},
reportid = {FZJ-2023-05145},
pages = {5125 - 5138},
year = {2023},
note = {Research funding: Bundesministerium für Bildung und
Forschung. Grant Numbers: BMBF 01ER0801, BMBF 01ER0803,
Deutsche Forschungsgemeinschaft. Grant Number: SO1336/1-1,
Horizon 2020 Framework Programme. Grant Number: 733280,
Kommission für Klinische Forschung, Technische Universität
München. Grant Numbers: 8700000474, 8765162},
abstract = {While animal models indicate altered brain dopaminergic
neurotransmission after premature birth, corresponding
evidence in humans is scarce due to missing molecular
imaging studies. To overcome this limitation, we studied
dopaminergic neurotransmission changes in human prematurity
indirectly by evaluating the spatial co-localization of
regional alterations in blood oxygenation fluctuations with
the distribution of adult dopaminergic neurotransmission.
The study cohort comprised 99 very premature-born
(<32 weeks of gestation and/or birth weight below
1500 g) and 107 full-term born young adults, being
assessed by resting-state functional MRI (rs-fMRI) and IQ
testing. Normative molecular imaging dopamine
neurotransmission maps were derived from independent healthy
control groups. We computed the co-localization of local
(rs-fMRI) activity alterations in premature-born adults with
respect to term-born individuals to different measures of
dopaminergic neurotransmission. We performed selectivity
analyses regarding other neuromodulatory systems and MRI
measures. In addition, we tested if the strength of the
co-localization is related to perinatal measures and IQ. We
found selectively altered co-localization of rs-fMRI
activity in the premature-born cohort with
dopamine-2/3-receptor availability in premature-born adults.
Alterations were specific for the dopaminergic system but
not for the used MRI measure. The strength of the
co-localization was negatively correlated with IQ. In line
with animal studies, our findings support the notion of
altered dopaminergic neurotransmission in prematurity which
is associated with cognitive performance.},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {37608591},
UT = {WOS:001052388000001},
doi = {10.1002/hbm.26451},
url = {https://juser.fz-juelich.de/record/1019092},
}