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@ARTICLE{Halfmann:1021445,
author = {Halfmann, Claas and Rüland, Thomas and Müller, Frank and
Jehasse, Kevin and Kampa, Björn M.},
title = {{E}lectrophysiological properties of layer 2/3 pyramidal
neurons in the primary visual cortex of a retinitis
pigmentosa mouse model (rd10)},
journal = {Frontiers in cellular neuroscience},
volume = {17},
issn = {1662-5102},
address = {Lausanne},
publisher = {Frontiers Research Foundation},
reportid = {FZJ-2024-00741},
pages = {1258773},
year = {2023},
note = {This study was funded by the Deutsche
Forschungsgemeinschaft(DFG, German Research
Foundation)−368482240/GRK2416 andGRK2610: Innoretvision.},
abstract = {Retinal degeneration is one of the main causes of visual
impairment and blindness. One group of retinal degenerative
diseases, leading to the loss of photoreceptors, is
collectively termed retinitis pigmentosa. In this group of
diseases, the remaining retina is largely spared from
initial cell death making retinal ganglion cells an
interesting target for vision restoration methods. However,
it is unknown how downstream brain areas, in particular the
visual cortex, are affected by the progression of blindness.
Visual deprivation studies have shown dramatic changes in
the electrophysiological properties of visual cortex
neurons, but changes on a cellular level in retinitis
pigmentosa have not been investigated yet. Therefore, we
used the rd10 mouse model to perform patch-clamp recordings
of pyramidal neurons in layer 2/3 of the primary visual
cortex to screen for potential changes in
electrophysiological properties resulting from retinal
degeneration. Compared to wild-type C57BL/6 mice, we only
found an increase in intrinsic excitability around the time
point of maximal retinal degeneration. In addition, we saw
an increase in the current amplitude of spontaneous putative
inhibitory events after a longer progression of retinal
degeneration. However, we did not observe a long-lasting
shift in excitability after prolonged retinal degeneration.
Together, our results provide evidence of an intact visual
cortex with promising potential for future therapeutic
strategies to restore vision.},
cin = {IBI-1 / INM-10},
ddc = {610},
cid = {I:(DE-Juel1)IBI-1-20200312 / I:(DE-Juel1)INM-10-20170113},
pnm = {5241 - Molecular Information Processing in Cellular Systems
(POF4-524) / GRK 2416 - GRK 2416: MultiSenses-MultiScales:
Neue Ansätze zur Aufklärung neuronaler multisensorischer
Integration (368482240) / GRK 2610 - GRK 2610: Innovative
Schnittstellen zur Retina für optimiertes künstliches
Sehen - InnoRetVision (424556709)},
pid = {G:(DE-HGF)POF4-5241 / G:(GEPRIS)368482240 /
G:(GEPRIS)424556709},
typ = {PUB:(DE-HGF)16},
doi = {10.3389/fncel.2023.1258773},
url = {https://juser.fz-juelich.de/record/1021445},
}