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@ARTICLE{Ibrahim:1021984,
author = {Ibrahim, Karim and Iturmendi-Sabater, Iciar and Vasishth,
Maya and Barron, Daniel S. and Guardavaccaro, MariaRose and
Funaro, Melissa C. and Holmes, Avram and McCarthy, Gregory
and Eickhoff, Simon B. and Sukhodolsky, Denis G.},
title = {{N}eural circuit disruptions of eye gaze processing in
autism spectrum disorder and schizophrenia: {A}n activation
likelihood estimation meta-analysis},
journal = {Schizophrenia research},
volume = {264},
issn = {0920-9964},
address = {Amsterdam [u.a.]},
publisher = {Elsevier Science},
reportid = {FZJ-2024-01123},
pages = {298 - 313},
year = {2024},
abstract = {BackgroundImpairment in social cognition, particularly eye
gaze processing, is a shared feature common to autism
spectrum disorder (ASD) and schizophrenia. However, it is
unclear if a convergent neural mechanism also underlies gaze
dysfunction in these conditions. The present study examined
whether this shared eye gaze phenotype is reflected in a
profile of convergent neurobiological dysfunction in ASD and
schizophrenia.MethodsActivation likelihood estimation (ALE)
meta-analyses were conducted on peak voxel coordinates
across the whole brain to identify spatial convergence.
Functional coactivation with regions emerging as significant
was assessed using meta-analytic connectivity modeling.
Functional decoding was also conducted.ResultsFifty-six
experiments (n = 30 with schizophrenia and n = 26 with ASD)
from 36 articles met inclusion criteria, which comprised 354
participants with ASD, 275 with schizophrenia and 613
healthy controls (1242 participants in total). In ASD,
aberrant activation was found in the left amygdala relative
to unaffected controls during gaze processing. In
schizophrenia, aberrant activation was found in the right
inferior frontal gyrus and supplementary motor area. Across
ASD and schizophrenia, aberrant activation was found in the
right inferior frontal gyrus and right fusiform gyrus during
gaze processing. Functional decoding mapped the left
amygdala to domains related to emotion processing and
cognition, the right inferior frontal gyrus to cognition and
perception, and the right fusiform gyrus to visual
perception, spatial cognition, and emotion perception. These
regions also showed meta-analytic connectivity to
frontoparietal and frontotemporal
circuitry.ConclusionAlterations in frontoparietal and
frontotemporal circuitry emerged as neural markers of gaze
impairments in ASD and schizophrenia. These findings have
implications for advancing transdiagnostic biomarkers to
inform targeted treatments for ASD and schizophrenia.
Previous article in issue},
cin = {INM-7},
ddc = {610},
cid = {I:(DE-Juel1)INM-7-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
pid = {G:(DE-HGF)POF4-5252},
typ = {PUB:(DE-HGF)16},
pubmed = {38215566},
UT = {WOS:001154995000001},
doi = {10.1016/j.schres.2023.12.003},
url = {https://juser.fz-juelich.de/record/1021984},
}