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@ARTICLE{Jiang:1024835,
      author       = {Jiang, Lin and Liang, Yi and Genon, Sarah and He, Runyang
                      and Yang, Qingqing and Yi, Chanlin and Yu, Liang and Yao,
                      Dezhong and Eickhoff, Simon B. and Dong, Debo and Li, Fali
                      and Xu, Peng},
      title        = {{S}patial–rhythmic network as a biomarker of familial
                      risk for psychotic bipolar disorder},
      journal      = {Nature Mental Health},
      volume       = {1},
      number       = {11},
      issn         = {2731-6076},
      address      = {London},
      publisher    = {Nature Publishing Group UK},
      reportid     = {FZJ-2024-02501},
      pages        = {887 - 899},
      year         = {2023},
      abstract     = {Neuronal rhythms with different temporospatial dynamics are
                      prominent signatures of brain operation. Yet, the
                      synchronous coupling across multiple rhythms and spatially
                      distributed subsystems, as well as its role in brain
                      cognition and disease, remains mysterious. Here we proposed
                      a conceptually new framework to construct the large-scale
                      spatial–rhythmic network (SRN) and apply it to
                      case–control P300 electroencephalogram datasets. Results
                      show that SRN configurations are essential substrates of
                      attentional allocation and immediate memory for healthy
                      controls (N = 235), yielding prominent inter-rhythmic
                      interactions between the δ-frontoparietal/δ-limbic network
                      and other rhythmic subnetworks during P300 generation.
                      Importantly, SRN deviances shared by patients with bipolar
                      disorder (N = 188) and their first-degree relatives
                      (N = 201) might be putative electrophysiological
                      biomarkers for clinical screening of individuals at high
                      familial risk of disease onset. The findings emphasize that
                      configurations of SRNs have a previously unrecognized role
                      in cognitive (dys)functions.},
      cin          = {INM-7},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-7-20090406},
      pnm          = {5252 - Brain Dysfunction and Plasticity (POF4-525)},
      pid          = {G:(DE-HGF)POF4-5252},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:001390101200002},
      doi          = {10.1038/s44220-023-00143-8},
      url          = {https://juser.fz-juelich.de/record/1024835},
}