% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Frieg:1025606,
author = {Frieg, Benedikt and Han, Mookyoung and Giller, Karin and
Dienemann, Christian and Riedel, Dietmar and Becker, Stefan
and Andreas, Loren B. and Griesinger, Christian and
Schröder, Gunnar F.},
title = {{C}ryo-{EM} structures of lipidic fibrils of amyloid-β
(1-40)},
journal = {Nature Communications},
volume = {15},
number = {1},
issn = {2041-1723},
address = {[London]},
publisher = {Nature Publishing Group UK},
reportid = {FZJ-2024-03000},
pages = {1297},
year = {2024},
abstract = {Alzheimer’s disease (AD) is a progressive and incurable
neurodegenerative disease characterized by the extracellular
deposition of amyloid plaques. Investigation into the
composition of these plaques revealed a high amount of
amyloid-β (Aβ) fibrils and a high concentration of lipids,
suggesting that fibrillipid interactions may also be
relevant for the pathogenesis of AD. Therefore, we grew
Aβ40 fibrils in the presence of lipid vesicles and
determined their structure by cryo-electron microscopy
(cryo-EM) to high resolution. The fold of the major
polymorph is similar to the structure of brain-seeded
fibrils reported previously. The majority of the lipids are
bound to the fibrils, as we show by cryo-EM and NMR
spectroscopy. This apparent lipid extraction from vesicles
observed here in vitro provides structural insights into
potentially disease-relevant fibril-lipid interactions.},
cin = {IBI-7},
ddc = {500},
cid = {I:(DE-Juel1)IBI-7-20200312},
pnm = {5244 - Information Processing in Neuronal Networks
(POF4-524)},
pid = {G:(DE-HGF)POF4-5244},
typ = {PUB:(DE-HGF)16},
pubmed = {38351005},
UT = {WOS:001161933400016},
doi = {10.1038/s41467-023-43822-x},
url = {https://juser.fz-juelich.de/record/1025606},
}