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@ARTICLE{Tscherpel:1030861,
author = {Tscherpel, Caroline and Mustin, Maike and Massimini,
Marcello and Paul, Theresa and Ziemann, Ulf and Fink, Gereon
R. and Grefkes, Christian},
title = {{L}ocal neuronal sleep after stroke: {T}he role of cortical
bistability in brain reorganization},
journal = {Brain stimulation},
volume = {17},
number = {4},
issn = {1935-861X},
address = {New York, NY [u.a.]},
publisher = {Elsevier},
reportid = {FZJ-2024-05470},
pages = {836 - 846},
year = {2024},
abstract = {Background: Acute cerebral ischemia triggers a number of
cellular mechanisms not only leading to excitotoxic cell
death but also to enhanced neuroplasticity, facilitating
neuronal reorganization and functional recovery.Objective:
Transferring these cellular mechanisms to neurophysiological
correlates adaptable to patients is crucial to promote
recovery post-stroke. The combination of TMS and EEG
constitutes a promising readout of neuronal network activity
in stroke patients.Methods: We used the combination of TMS
and EEG to investigate the development of local signal
processing and global network alterations in 40 stroke
patients with motor deficits alongside neural reorganization
from the acute to the chronic phase.Results: We show that
the TMS-EEG response reflects information about
reorganization and signal alterations associated with
persistent motor deficits throughout the entire post-stroke
period. In the early post-stroke phase and in a subgroup of
patients with severe motor deficits, TMS applied to the
lesioned motor cortex evoked a sleep-like slow wave response
associated with a cortical off-period, a manifestation of
cortical bistability, as well as a rapid disruption of the
TMS-induced formation of causal network effects.
Mechanistically, these phenomena were linked to lesions
affecting ascending activating brainstem fibers. Of note,
slow waves invariably vanished in the chronic phase, but
were highly indicative of a poor functional
outcome.Conclusion: In summary, we found evidence that
transient effects of sleep-like slow waves and cortical
bistability within ipsilesional M1 resulting in excessive
inhibition may interfere with functional reorganization,
leading to a less favorable functional outcome post-stroke,
pointing to a new therapeutic target to improve recovery of
function.Keywords: Neuromodulation; Neurorehabilitation;
Plasticity; Sleep-like cortical off-period; Slow waves;
Stroke lesion; TMS-EEG.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {5252 - Brain Dysfunction and Plasticity (POF4-525) / DFG
project 431549029 - SFB 1451: Schlüsselmechanismen normaler
und krankheitsbedingt gestörter motorischer Kontrolle
(431549029)},
pid = {G:(DE-HGF)POF4-5252 / G:(GEPRIS)431549029},
typ = {PUB:(DE-HGF)16},
pubmed = {39019396},
UT = {WOS:001274661900001},
doi = {10.1016/j.brs.2024.07.008},
url = {https://juser.fz-juelich.de/record/1030861},
}