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@ARTICLE{Garibotto:10795,
      author       = {Garibotto, V. and Borroni, B. and Agosti, C. and Premi, E.
                      and Alberici, A. and Eickhoff, S. B. and Brambati, S.M. and
                      Bellelli, G. and Gasparotti, R. and Perani, D. and Padovani,
                      A.},
      title        = {{S}ubcortical and deep cortical atrophy in {F}rontotemporal
                      {L}obar {D}egeneration},
      journal      = {Neurobiology of aging},
      volume       = {32},
      issn         = {0197-4580},
      address      = {Amsterdam [u.a.]},
      publisher    = {Elsevier Science},
      reportid     = {PreJuSER-10795},
      pages        = {875 - 884},
      year         = {2011},
      note         = {This work was financially supported by EULO (Ente
                      Universitario Lombardia Orientale), and DIMI (Diagnostic
                      Molecular Imaging). Sixth European Program, Project No:
                      LSHB-CT-2005-512146.},
      abstract     = {Though neuroimaging, pathology and pathophysiology suggest
                      a subcortical and deep cortical involvement in
                      Frontotemporal Lobar Degeneration (FTLD), no studies have
                      comprehensively assessed the associated gray matter (GM)
                      volume changes. We measured caudate, putamen, thalamus, and
                      amygdala GM volume using probabilistic a-priori regions of
                      interest (ROIs) in 53 early FTLD patients (38 behavioral
                      variant FTD [bvFTD], 9 Semantic Dementia [SD], 6 Progressive
                      Non-Fluent Aphasia [PNFA]), and 25 age-matched healthy
                      controls (HC). ANOVA showed significant (P<0.001) main
                      effect of diagnosis, and significant interactions for
                      diagnosis and region, and diagnosis and hemisphere. Post-hoc
                      comparisons with HC showed bilateral GM atrophy in the
                      caudate, putamen and thalamus, in bvFTD; a left-confined GM
                      reduction in the amygdala in SD; and bilateral GM atrophy in
                      the caudate and thalamus, and left-sided GM reduction in the
                      putamen and amygdala in PNFA. Correlation analyses suggested
                      an association between GM volumes and language, psychomotor
                      speed and behavioral disturbances. This study showed a
                      widespread involvement of subcortical and deep cortical GM
                      in early FTLD with patterns specific for clinical entity.
                      (C) 2009 Elsevier Inc. All rights reserved.},
      keywords     = {J (WoSType)},
      cin          = {INM-2},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-2-20090406},
      pnm          = {Funktion und Dysfunktion des Nervensystems (FUEK409) /
                      89571 - Connectivity and Activity (POF2-89571)},
      pid          = {G:(DE-Juel1)FUEK409 / G:(DE-HGF)POF2-89571},
      shelfmark    = {Geriatrics $\&$ Gerontology / Neurosciences},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000289957000012},
      doi          = {10.1016/j.neurobiologaing.2009.05.004},
      url          = {https://juser.fz-juelich.de/record/10795},
}