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@ARTICLE{Herz:139805,
      author       = {Herz, D. M. and Florin, E. and Christensen, M. S. and Reck,
                      C. and Barbe, Michael and Tscheuschler, M. K. and
                      Tittgemeyer, M. and Siebner, H. R. and Timmermann, L.},
      title        = {{D}opamine {R}eplacement {M}odulates {O}scillatory
                      {C}oupling {B}etween {P}remotor and {M}otor {C}ortical
                      {A}reas in {P}arkinson's {D}isease.},
      journal      = {Cerebral cortex},
      volume       = {24},
      number       = {11},
      issn         = {1460-2199},
      address      = {Oxford},
      publisher    = {Oxford Univ. Press},
      reportid     = {FZJ-2013-05776},
      pages        = {2873-2883},
      year         = {2014},
      abstract     = {Efficient neural communication between premotor and motor
                      cortical areas is critical for manual motor control. Here,
                      we used high-density electroencephalography to study
                      cortical connectivity in patients with Parkinson's disease
                      (PD) and age-matched healthy controls while they performed
                      repetitive movements of the right index finger at maximal
                      repetition rate. Multiple source beamformer analysis and
                      dynamic causal modeling were used to assess oscillatory
                      coupling between the lateral premotor cortex (lPM),
                      supplementary motor area (SMA), and primary motor cortex
                      (M1) in the contralateral hemisphere. Elderly healthy
                      controls showed task-related modulation in connections from
                      lPM to SMA and M1, mainly within the γ-band (>30 Hz).
                      Nonmedicated PD patients also showed task-related γ-γ
                      coupling from lPM to M1, but γ coupling from lPM to SMA was
                      absent. Levodopa reinstated physiological γ-γ coupling
                      from lPM to SMA and significantly strengthened coupling in
                      the feedback connection from M1 to lPM expressed as β-β as
                      well as θ-β coupling. Enhancement in cross-frequency θ-β
                      coupling from M1 to lPM was correlated with levodopa-induced
                      improvement in motor function. The results show that PD is
                      associated with an altered neural communication between
                      premotor and motor cortical areas, which can be modulated by
                      dopamine replacement.},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {333 - Pathophysiological Mechanisms of Neurological and
                      Psychiatric Diseases (POF2-333) / 89572 - (Dys-)function and
                      Plasticity (POF2-89572)},
      pid          = {G:(DE-HGF)POF2-333 / G:(DE-HGF)POF2-89572},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000344646200006},
      doi          = {10.1093/cercor/bht140},
      url          = {https://juser.fz-juelich.de/record/139805},
}