% IMPORTANT: The following is UTF-8 encoded. This means that in the presence % of non-ASCII characters, it will not work with BibTeX 0.99 or older. % Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or % “biber”. @ARTICLE{Amin:140000, author = {Amin, Ehsan and Dubey, Badri Nath and Zhang, Si-Cai and Gremer, Lothar and Dvorsky, Radovan and Moll, Jens M. and Taha, Mohamed S. and Nagel-Steger, Luitgard and Piekorz, Roland P. and Somlyo, Avril V. and Ahmadian, Mohammad R.}, title = {{R}ho-kinase: regulation, (dys)function, and inhibition}, journal = {Biological chemistry}, volume = {394}, number = {11}, issn = {1437-4315}, address = {Berlin [u.a.]}, publisher = {de Gruyter}, reportid = {FZJ-2013-05967}, pages = {1399-1410}, year = {2013}, abstract = {In a variety of normal and pathological cell types, Rho-kinases I and II (ROCKI/II) play a pivotal role in the organization of the nonmuscle and smooth muscle cytoskeleton and adhesion plaques as well as in the regulation of transcription factors. Thus, ROCKI/II activity regulates cellular contraction, motility, morphology, polarity, cell division, and gene expression. Emerging evidence suggests that dysregulation of the Rho-ROCK pathways at different stages is linked to cardiovascular, metabolic, and neurodegenerative diseases as well as cancer. This review focuses on the current status of understanding the multiple functions of Rho-ROCK signaling pathways and various modes of regulation of Rho-ROCK activity, thereby orchestrating a concerted functional response.}, cin = {ICS-6}, ddc = {540}, cid = {I:(DE-Juel1)ICS-6-20110106}, pnm = {452 - Structural Biology (POF2-452)}, pid = {G:(DE-HGF)POF2-452}, typ = {PUB:(DE-HGF)16}, UT = {WOS:000325717100005}, doi = {10.1515/hsz-2013-0181}, url = {https://juser.fz-juelich.de/record/140000}, }