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@ARTICLE{Kpper:14490,
author = {Küpper, K. and Lang, N. and Möhl, C. and Kirchgeßner, N.
and Born, S. and Goldmann, W.H. and Hoffmann, B. and Merkel,
R.},
title = {{T}yrosine phosphorylation of vinculin at position 1065
modifies focal adhesion dynamics and cell tractions},
journal = {Biochemical and biophysical research communications},
volume = {399},
issn = {0006-291X},
address = {Orlando, Fla.},
publisher = {Academic Press},
reportid = {PreJuSER-14490},
pages = {560 - 564},
year = {2010},
note = {We thank Dr. E.D. Adamson for providing the MEF cells and
Dr. B. Fabry for stimulating discussions. We are also
indebted to the entire team of IBN 4 for their interlectual
input. This work was supported by grants from
Bayerisch-Franzosisches Hochschulzentrum, Deutscher
Akademischer Austausch Dienst, Bavaria California Technology
Center, Deutsche Forschungsgemeinschaft (G0598/13-1), and
Bundesministerium fur Bildung und Forschung (Program
0315501).},
abstract = {Focal adhesions (FAs) connect the cellular actin
cytoskeleton via integrin with the extracellular matrix.
They comprise of many structural and signaling proteins
which are highly dynamic, well regulated, and responsible
for the sensing of physical properties from the environment.
Vinculin is a protein that incorporates all these functions.
Here, we investigated the phosphorylation of Y1065 in the
activation/regulation of vinculin. We used different
vinculin mutants mimicking either a permanently activated or
inhibited phosphorylation site at position 1065. Using these
mutants, we determined their influence on the exchange
dynamics and cell forces using fluorescence recovery after
photobleaching and traction microscopy. The results indicate
that phosphorylation at Y1065 significantly increases the
amount of freely exchanging vinculin within FAs whereas
inhibition of this phosphorylation site leads to an
uncontrolled exchange of vinculin and reduced adhesive cell
forces. In conclusion, we show that phosphorylation on
position Y1065 is essential for accurate incorporation of
vinculin into FAs and mechanical behavior of cells.},
keywords = {Animals / Focal Adhesions: genetics / Focal Adhesions:
metabolism / Mice / Mice, Knockout / Mutation /
Phosphorylation / Tyrosine: genetics / Tyrosine: metabolism
/ Vinculin: genetics / Vinculin: metabolism / Vinculin (NLM
Chemicals) / Tyrosine (NLM Chemicals) / J (WoSType)},
cin = {IBN-4},
ddc = {570},
cid = {I:(DE-Juel1)VDB802},
pnm = {BioSoft: Makromolekulare Systeme und biologische
Informationsverarbeitung},
pid = {G:(DE-Juel1)FUEK505},
shelfmark = {Biochemistry $\&$ Molecular Biology / Biophysics},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:20678470},
UT = {WOS:000281840200017},
doi = {10.1016/j.bbrc.2010.07.110},
url = {https://juser.fz-juelich.de/record/14490},
}