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@ARTICLE{Mirecka:153349,
author = {Mirecka, E. A and Shaykhalishahi, H. and Gauhar, A. and
Akgül, S. and Lecher, Justin and Willbold, Dieter and
Stoldt, Matthias and Hoyer, W.},
title = {{S}equestration of a β-{H}airpin for {C}ontrol of
α-{S}ynuclein {A}ggregation},
journal = {Angewandte Chemie / International edition},
volume = {53},
number = {16},
issn = {0570-0833},
address = {Weinheim},
publisher = {Wiley-VCH},
reportid = {FZJ-2014-02981},
pages = {4227-4230},
year = {2014},
abstract = {The misfolding and aggregation of the protein α-synuclein
(α-syn), which results in the formation of amyloid fibrils,
is involved in the pathogenesis of Parkinson's disease and
other synucleinopathies. The emergence of amyloid toxicity
is associated with the formation of partially folded
aggregation intermediates. Here, we engineered a class of
binding proteins termed β-wrapins (β-wrap proteins) with
affinity for α-synuclein (α-syn). The NMR structure of an
α-syn:β-wrapin complex reveals a β-hairpin of α-syn
comprising the sequence region α-syn(37-54). The β-wrapin
inhibits α-syn aggregation and toxicity at
substoichiometric concentrations, demonstrating that it
interferes with the nucleation of aggregation.© 2014
WILEY-VCH Verlag GmbH $\&$ Co. KGaA, Weinheim.},
cin = {ICS-6},
ddc = {540},
cid = {I:(DE-Juel1)ICS-6-20110106},
pnm = {452 - Structural Biology (POF2-452)},
pid = {G:(DE-HGF)POF2-452},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:24623599},
UT = {WOS:000338968700022},
doi = {10.1002/anie.201309001},
url = {https://juser.fz-juelich.de/record/153349},
}