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@INPROCEEDINGS{Moos:153470,
      author       = {Moos, K. and Weidner, R. and Vossel, S. and Zimmermann, E.
                      and Dyrholm, M. and Fink, Gereon Rudolf},
      title        = {{S}triatal involvement in visual encoding},
      journal      = {Klinische Neurophysiologie},
      volume       = {45},
      number       = {01},
      issn         = {1439-4081},
      address      = {Stuttgart [u.a.]},
      publisher    = {Thieme},
      reportid     = {FZJ-2014-03068},
      pages        = {s-0034-1371195},
      year         = {2014},
      abstract     = {Klin Neurophysiol 2014; 45 - V16DOI:
                      10.1055/s-0034-1371195Striatal involvement in visual
                      encodingK Moos 1, 2, R Weidner 1, S Vossel 1, E Zimmermann
                      1, M Dyrholm 3, GR Fink 1, 2 1Forschungszentrum Jülich,
                      Cognitive Neuroscience, Jülich, Deutschland 2Uniklinik
                      Köln, Klinik und Poliklinik für Neurologie, Köln,
                      Deutschland 3Universität Kopenhagen, Institut für
                      Psychologie, Kopenhagen, Dänemark KongressbeitragA capacity
                      limited system, such as the human brain, needs effective
                      strategies to deal with large amounts of incoming
                      information. The theory of visual attention (TVA) allows for
                      formal quantification of specific processes related to
                      selection and recognition of visual information. In the
                      present model-based fMRI study, parameters derived from the
                      TVA-framework were used to determine trial-by-trial changes
                      in the distribution of attention and to relate these to
                      specific cortical attentional networks. Trial-by-trial
                      changes in the single trial likelihood (STL), an inferred
                      variable representing an element's probability of being
                      encoded into the visual short term memory (VSTM) and thus
                      consciously perceived, were accompanied by changes in
                      activity in the bilateral putamen and a right-lateralized
                      network involving the inferior parietal lobule. Moreover,
                      attentional networks were activated by additional display
                      elements: With higher competition between relevant display
                      elements (i.e., when two targets rather than one target and
                      a distractor were presented), a bilateral dorsal network
                      comprising the frontal eye fields and superior parietal
                      regions was more active. Bilateral temporo-parietal junction
                      and superior frontal regions showed higher activity in the
                      reversed contrast, reflecting distractor-related filtering
                      processes.We demonstrate that basal ganglia, i.e., the
                      bilateral striatum, are critically involved in the encoding
                      of visual information into the VSTM to a perceptual level of
                      processing. Moreover, we specify the function of the dorsal
                      attention network in resolving competition between equally
                      important display elements, whereas a bilateral ventral
                      network processes imbalances in the distribution of
                      attentional resources.},
      month         = {Mar},
      date          = {2014-03-19},
      organization  = {58. Jahrestagung der Deutschen
                       Gesellschaft für Klinische
                       Neurophysiologie und Funktionelle
                       Bildgebung (DGKN), Berlin (Germany), 19
                       Mar 2014 - 22 Mar 2014},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {333 - Pathophysiological Mechanisms of Neurological and
                      Psychiatric Diseases (POF2-333) / 89572 - (Dys-)function and
                      Plasticity (POF2-89572)},
      pid          = {G:(DE-HGF)POF2-333 / G:(DE-HGF)POF2-89572},
      typ          = {PUB:(DE-HGF)8 / PUB:(DE-HGF)16},
      doi          = {10.1055/s-0034-1371195},
      url          = {https://juser.fz-juelich.de/record/153470},
}