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@ARTICLE{Langen:155422,
      author       = {Langen, K. J. and Tonn, J. C. and Weller, M. and Galldiks,
                      N.},
      title        = {{L}etter to the {E}ditor: “{T}he role of imaging in the
                      management of progressive glioblastoma. {A} systematic
                      review and evidence-based clinical practice guideline”
                      [{J} {N}eurooncol 2014; 118:435–460]},
      journal      = {Journal of neuro-oncology},
      volume       = {120},
      number       = {3},
      issn         = {0167-594x},
      address      = {Dordrecht [u.a.]},
      publisher    = {Springer Science + Business Media B.V},
      reportid     = {FZJ-2014-04588},
      pages        = {665-666},
      year         = {2014},
      abstract     = {o the Editor,We have read with interest the review by Ryken
                      et al. about the role of imaging in the management of
                      progressive glioblastoma [1]. In general, we agree with this
                      review but we cannot support the opinion that the routine
                      use of Positron-Emission-Tomography (PET) to identify
                      progression of glioblastoma is not recommendable.The authors
                      have considered PET using the amino acid tracer
                      11C-methyl-l-methionine (MET), but the use of MET is limited
                      to PET centers with an on-site cyclotron due the short
                      half-life of 11C (20.4 min). In recent years, the clinical
                      application of 18F-labeled amino acids such as
                      O-(2-18F-fluoroethyl)-l-tyrosine (FET) or
                      3,4-dihydroxy-6-[18F]fluoro-l-phenylalanine (FDOPA) has
                      spread considerably due to the logistical advantages of the
                      18F label (half-life, 109.8 min) [2]. FET can be produced
                      with high yields similar to the widely used FDG and
                      distributed in a satellite concept [3]. In Europe, MET PET
                      has been replaced in many centers by the more convenie ...},
      cin          = {INM-3 / INM-4},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406 / I:(DE-Juel1)INM-4-20090406},
      pnm          = {333 - Pathophysiological Mechanisms of Neurological and
                      Psychiatric Diseases (POF2-333) / 89572 - (Dys-)function and
                      Plasticity (POF2-89572)},
      pid          = {G:(DE-HGF)POF2-333 / G:(DE-HGF)POF2-89572},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000345286700028},
      doi          = {10.1007/s11060-014-1594-z},
      url          = {https://juser.fz-juelich.de/record/155422},
}