Home > Publications database > Primary intracranial plasma cell granuloma responsive to rituximab > print |
001 | 172851 | ||
005 | 20210129214522.0 | ||
024 | 7 | _ | |a 10.1212/WNL.0000000000000799 |2 doi |
024 | 7 | _ | |a 0028-3878 |2 ISSN |
024 | 7 | _ | |a 1526-632X |2 ISSN |
024 | 7 | _ | |a WOS:000342230700016 |2 WOS |
037 | _ | _ | |a FZJ-2014-06285 |
082 | _ | _ | |a 610 |
100 | 1 | _ | |a Schneider, C. |0 P:(DE-HGF)0 |b 0 |e Corresponding Author |
245 | _ | _ | |a Primary intracranial plasma cell granuloma responsive to rituximab |
260 | _ | _ | |a Hagerstown, Md. |c 2014 |b Lippincott Williams & Wilkins |
336 | 7 | _ | |a Journal Article |b journal |m journal |0 PUB:(DE-HGF)16 |s 1437391731_27741 |2 PUB:(DE-HGF) |
336 | 7 | _ | |a Output Types/Journal article |2 DataCite |
336 | 7 | _ | |a Journal Article |0 0 |2 EndNote |
336 | 7 | _ | |a ARTICLE |2 BibTeX |
336 | 7 | _ | |a JOURNAL_ARTICLE |2 ORCID |
336 | 7 | _ | |a article |2 DRIVER |
520 | _ | _ | |a Plasma cell granuloma (PCG) is a sporadically occurring tumor-like mass that is histologically characterized by a non-neoplastic proliferation of inflammatory cells. The predominant cell types in PCG are polyclonal plasma cells and their precursors, including a substantial amount of mature B cells. |
536 | _ | _ | |a 333 - Pathophysiological Mechanisms of Neurological and Psychiatric Diseases (POF2-333) |0 G:(DE-HGF)POF2-333 |c POF2-333 |f POF II |x 0 |
588 | _ | _ | |a Dataset connected to CrossRef, juser.fz-juelich.de |
700 | 1 | _ | |a Henning, T. D. |0 P:(DE-HGF)0 |b 1 |
700 | 1 | _ | |a Fink, G. R. |0 P:(DE-Juel1)131720 |b 2 |u fzj |
700 | 1 | _ | |a Schroeter, M. |0 P:(DE-HGF)0 |b 3 |
700 | 1 | _ | |a Lehmann, H. C. |0 P:(DE-HGF)0 |b 4 |
773 | _ | _ | |a 10.1212/WNL.0000000000000799 |g Vol. 83, no. 12, p. 1119 - 1120 |0 PERI:(DE-600)1491874-2 |n 12 |p 1119 - 1120 |t Neurology |v 83 |y 2014 |x 1526-632X |
909 | C | O | |o oai:juser.fz-juelich.de:172851 |p VDB |
910 | 1 | _ | |a Forschungszentrum Jülich GmbH |0 I:(DE-588b)5008462-8 |k FZJ |b 2 |6 P:(DE-Juel1)131720 |
913 | 2 | _ | |a DE-HGF |b POF III |l Key Technologies |1 G:(DE-HGF)POF3-570 |0 G:(DE-HGF)POF3-572 |2 G:(DE-HGF)POF3-500 |v Decoding the Human Brain |x 0 |
913 | 1 | _ | |a DE-HGF |b Gesundheit |l Funktion und Dysfunktion des Nervensystems |1 G:(DE-HGF)POF2-330 |0 G:(DE-HGF)POF2-333 |2 G:(DE-HGF)POF2-300 |v Pathophysiological Mechanisms of Neurological and Psychiatric Diseases |x 0 |4 G:(DE-HGF)POF |3 G:(DE-HGF)POF2 |
914 | 1 | _ | |y 2014 |
915 | _ | _ | |a Allianz-Lizenz |0 StatID:(DE-HGF)0410 |2 StatID |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0300 |2 StatID |b Medline |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0310 |2 StatID |b NCBI Molecular Biology Database |
915 | _ | _ | |a JCR |0 StatID:(DE-HGF)0100 |2 StatID |
915 | _ | _ | |a IF >= 5 |0 StatID:(DE-HGF)9905 |2 StatID |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0200 |2 StatID |b SCOPUS |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0199 |2 StatID |b Thomson Reuters Master Journal List |
915 | _ | _ | |a WoS |0 StatID:(DE-HGF)0110 |2 StatID |b Science Citation Index |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0150 |2 StatID |b Web of Science Core Collection |
915 | _ | _ | |a WoS |0 StatID:(DE-HGF)0111 |2 StatID |b Science Citation Index Expanded |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)1110 |2 StatID |b Current Contents - Clinical Medicine |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)1030 |2 StatID |b Current Contents - Life Sciences |
915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)1050 |2 StatID |b BIOSIS Previews |
920 | 1 | _ | |0 I:(DE-Juel1)INM-3-20090406 |k INM-3 |l Kognitive Neurowissenschaften |x 0 |
980 | _ | _ | |a journal |
980 | _ | _ | |a VDB |
980 | _ | _ | |a I:(DE-Juel1)INM-3-20090406 |
980 | _ | _ | |a UNRESTRICTED |
Library | Collection | CLSMajor | CLSMinor | Language | Author |
---|