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@ARTICLE{Maier:187642,
author = {Maier, F. and Ellereit, A. L. and Eggers, C. and Lewis, C.
J. and Pelzer, E. and Kalbe, E. and Ernstmann, N. and
Prigatano, G. P. and Fink, Gereon Rudolf and Timmermann, L.},
title = {{D}evelopment and psychometric evaluation of a scale to
measure impaired self-awareness of hyper- and hypokinetic
movements in {P}arkinson’s disease},
journal = {Journal of the International Neuropsychological Society},
volume = {21},
number = {3},
issn = {1355-6177},
address = {Cambridge},
publisher = {Cambridge Univ. Press},
reportid = {FZJ-2015-01268},
pages = {221-230},
year = {2015},
abstract = {Objective: Patients with Parkinson’s disease (PD) can
show impaired self-awareness of motor deficits (ISAm). We
developed a new scale that measures ISAm severity of hyper-
and hypokinetic movements in PD during medication on state
and defined its psychometric criteria. Method: Included were
104 right-handed, non-depressed, non-demented patients.
Concerning ISAm, 38 motor symptoms were assessed using seven
tasks, which were performed and self-rated concerning
presence of deficit (yes/no) by all patients. The whole
procedure was videotaped. Motor symptoms were then evaluated
by two independent experts, blinded for patient’s ratings,
concerning presence, awareness of deficit, and severity.
Exploratory principal component analysis (promax rotation)
was applied to reduce items. Principal axis factoring was
conducted to extract factors. Reliability was examined
regarding internal consistency, split-half reliability, and
interrater reliability. Validity was verified by applying
two additional measures of ISAm. Results: Of the initial 38
symptoms, 15 remained, assessed in five motor tasks and
merged to a total severity score. Factor analysis resulted
in a four factor solution (dyskinesia, resting tremor right
hand, resting tremor left hand, bradykinesia). For all
subscales and the total score, measures of reliability
(values 0.64–0.89) and validity (effect sizes>0.3) were
satisfactory. Descriptive results showed that $66\%$ of
patients had signs of ISAm (median 2, range 0–15), with
ISAm being most distinct for dyskinesia. Conclusions: We
provide the first validation of a test for ISAm in PD. Using
this instrument, future studies can further analyze the
pathophysiology of ISAm, the psychosocial sequelae,
therapeutic strategies and compliance with therapy. (JINS,
2015, 21, 1–10)},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000354027000005},
pubmed = {pmid:25687696},
doi = {10.1017/S1355617715000107},
url = {https://juser.fz-juelich.de/record/187642},
}