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@ARTICLE{New:187770,
      author       = {New, Anneliese B. and Robin, Donald A and Parkinson, Amy L
                      and Eickhoff, Claudia R and Reetz, Kathrin and Hoffstaedter,
                      Felix and Mathys, Christian and Südmeyer, Martin and
                      Grefkes, Christian and Larson, Charles R and Ramig, Loraine
                      O and Fox, Peter T and Eickhoff, Simon},
      title        = {{T}he intrinsic resting state voice network in
                      {P}arkinson's disease.},
      journal      = {Human brain mapping},
      volume       = {36},
      number       = {5},
      issn         = {1065-9471},
      address      = {New York, NY},
      publisher    = {Wiley-Liss},
      reportid     = {FZJ-2015-01352},
      pages        = {1951–1962},
      year         = {2015},
      abstract     = {Over 90 percent of patients with Parkinson's disease
                      experience speech-motor impairment, namely, hypokinetic
                      dysarthria characterized by reduced pitch and loudness.
                      Resting-state functional connectivity analysis of blood
                      oxygen level-dependent functional magnetic resonance imaging
                      is a useful measure of intrinsic neural functioning. We
                      utilized resting-state functional connectivity modeling to
                      analyze the intrinsic connectivity in patients with
                      Parkinson's disease within a vocalization network defined by
                      a previous meta-analysis of speech (Brown et al., 2009).
                      Functional connectivity of this network was assessed in 56
                      patients with Parkinson's disease and 56 gender-, age-, and
                      movement-matched healthy controls. We also had item 5 and 18
                      of the UPDRS, and the PDQ-39 Communication subscale
                      available for correlation with the voice network
                      connectivity strength in patients. The within-group analyses
                      of connectivity patterns demonstrated a lack of
                      subcortical–cortical connectivity in patients with
                      Parkinson's disease. At the cortical level, we found robust
                      (homotopic) interhemispheric connectivity but only
                      inconsistent evidence for many intrahemispheric connections.
                      When directly contrasted to the control group, we found a
                      significant reduction of connections between the left
                      thalamus and putamen, and cortical motor areas, as well as
                      reduced right superior temporal gyrus connectivity.
                      Furthermore, most symptom measures correlated with right
                      putamen, left cerebellum, left superior temporal gyrus,
                      right premotor, and left Rolandic operculum connectivity in
                      the voice network. The results reflect the importance of
                      (right) subcortical nodes and the superior temporal gyrus in
                      Parkinson's disease, enhancing our understanding of the
                      neurobiological underpinnings of vocalization impairment in
                      Parkinson's disease},
      cin          = {INM-1 / INM-4 / INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-1-20090406 / I:(DE-Juel1)INM-4-20090406 /
                      I:(DE-Juel1)INM-3-20090406},
      pnm          = {571 - Connectivity and Activity (POF3-571)},
      pid          = {G:(DE-HGF)POF3-571},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:25627959},
      UT           = {WOS:000353068300023},
      doi          = {10.1002/hbm.22748},
      url          = {https://juser.fz-juelich.de/record/187770},
}