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000189359 005__ 20210129215344.0
000189359 0247_ $$2doi$$a10.1038/nn.3922
000189359 0247_ $$2ISSN$$a1097-6256
000189359 0247_ $$2ISSN$$a1471-003X
000189359 0247_ $$2ISSN$$a1471-0048
000189359 0247_ $$2ISSN$$a1546-1726
000189359 0247_ $$2WOS$$aWOS:000348631800010
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000189359 0247_ $$2pmid$$apmid:25599223
000189359 037__ $$aFZJ-2015-02537
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000189359 1001_ $$aO'Dushlaine, Colm$$b0
000189359 245__ $$aPsychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways
000189359 260__ $$aNew York, NY$$bNature America$$c2015
000189359 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1428931228_30909
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000189359 3367_ $$2BibTeX$$aARTICLE
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000189359 3367_ $$2DRIVER$$aarticle
000189359 520__ $$aGenome-wide association studies (GWAS) of psychiatric disorders have identified multiple genetic associations with such disorders, but better methods are needed to derive the underlying biological mechanisms that these signals indicate. We sought to identify biological pathways in GWAS data from over 60,000 participants from the Psychiatric Genomics Consortium. We developed an analysis framework to rank pathways that requires only summary statistics. We combined this score across disorders to find common pathways across three adult psychiatric disorders: schizophrenia, major depression and bipolar disorder. Histone methylation processes showed the strongest association, and we also found statistically significant evidence for associations with multiple immune and neuronal signaling pathways and with the postsynaptic density. Our study indicates that risk variants for psychiatric disorders aggregate in particular biological pathways and that these pathways are frequently shared between disorders. Our results confirm known mechanisms and suggest several novel insights into the etiology of psychiatric disorders.
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000189359 7001_ $$aZhao, Zhaoming$$b369
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000189359 7001_ $$0P:(DE-HGF)0$$aBreen, Gerome$$b374$$eCorresponding Author
000189359 773__ $$0PERI:(DE-600)1494955-6$$a10.1038/nn.3922$$gVol. 18, no. 2, p. 199 - 209$$n2$$p199 - 209$$tNature neuroscience$$v18$$x1097-6256$$y2015
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