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@ARTICLE{Musiani:201310,
author = {Musiani, Francesco and Ippoliti, Emiliano and Micheletti,
Cristian and Carloni, Paolo and Ciurli, Stefano},
title = {{C}onformational {F}luctuations of {U}re{G}, an
{I}ntrinsically {D}isordered {E}nzyme},
journal = {Biochemistry},
volume = {52},
number = {17},
issn = {1520-4995},
address = {Columbus, Ohio},
publisher = {American Chemical Society},
reportid = {FZJ-2015-03615},
pages = {2949 - 2954},
year = {2013},
abstract = {UreG proteins are small GTP binding (G) proteins that
catalyze the hydrolysis of GTP necessary for the maturation
of urease, a virulence factor in bacterial pathogenesis.
UreG proteins are the first documented cases of
intrinsically disordered enzymes. The comprehension of the
dynamics of folding−unfolding events occurring in this
protein could shed light on the enzymatic mechanism of UreG.
Here, we used the recently developed replica exchange with
solute tempering (REST2) computational methodology to
explore the conformational space of UreG from Helicobacter
pylori (HpUreG) and to identify its structural fluctuations.
The same simulation and analysis protocol has been applied
to HypB from Methanocaldococcus jannaschii (MjHypB), which
is closely related to UreG in both sequence and function,
even though it is not intrinsically disordered. A comparison
of the two systems reveals that both HpUreG and MjHypB
feature a substantial rigidity of the protein regions
involved in catalysis, justifying its residual catalytic
activity. On the other hand, HpUreG tends to unfold more
than MjHypB in portions involved in protein−protein
interactions with metallochaperones necessary for the
formation of multiprotein complexes known to be involved in
urease activation.},
cin = {GRS / IAS-5},
ddc = {570},
cid = {I:(DE-Juel1)GRS-20100316 / I:(DE-Juel1)IAS-5-20120330},
pnm = {899 - ohne Topic (POF2-899)},
pid = {G:(DE-HGF)POF2-899},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000318333100010},
doi = {10.1021/bi4001744},
url = {https://juser.fz-juelich.de/record/201310},
}