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@ARTICLE{vonPapen:201391,
author = {von Papen, Mitra and Fisse, Mirabell and Sarfeld,
Anna-Sophia and Fink, Gereon R. and Nowak, Dennis A.},
title = {{T}he effects of 1 {H}z r{TMS} preconditioned by t{DCS} on
gait kinematics in {P}arkinson’s disease},
journal = {Journal of neural transmission},
volume = {121},
number = {7},
issn = {1435-1463},
address = {Wien [u.a.]},
publisher = {Springer},
reportid = {FZJ-2015-03686},
pages = {743 - 754},
year = {2014},
abstract = {Hypokinetic gait is a common and very disabling symptom of
Parkinson’s disease (PD). Repetitive transcranial magnetic
stimulation (rTMS) over the motor cortex has been used with
variable effectiveness to treat hypokinesia in PD.
Preconditioning rTMS by transcranial direct current
stimulation (tDCS) may enhance its effectiveness to treat
hypokinetic gait in PD. Three-dimensional kinematic gait
analysis was performed (1) prior to, (2) immediately after
and (3) 30 min after low-frequency rTMS (1 Hz, 900 pulses,
80 $\%$ of resting motor threshold) over M1 contralateral to
the more affected body side preconditioned by (1) cathodal,
(2) anodal or (3) sham tDCS (amperage: 1 mA, duration: 10
min) in ten subjects with PD (7 females, mean age 63 ± 9
years) and ten healthy subjects (four females, mean age 50
± 11 years). The effects of tDCS-preconditioned rTMS on
gait kinematics were assessed by the following parameters:
number of steps, step length, stride length, double support
time, cadence, swing and stance phases. Our data suggest a
bilateral improvement of hypokinetic gait in PD after 1 Hz
rTMS over M1 of the more affected body side preceded by
anodal tDCS. In contrast, 1 Hz rTMS alone (preceded by sham
tDCS) and 1 Hz rTMS preceded by cathodal tDCS were
ineffective to improve gait kinematics in PD. In healthy
subjects, gait kinematics was unaffected by either
intervention. Preconditioning motor cortex rTMS by tDCS is a
promising approach to treat hypokinetic gait in PD.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {333 - Pathophysiological Mechanisms of Neurological and
Psychiatric Diseases (POF2-333)},
pid = {G:(DE-HGF)POF2-333},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000338276800007},
pubmed = {pmid:24562404},
doi = {10.1007/s00702-014-1178-2},
url = {https://juser.fz-juelich.de/record/201391},
}