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@ARTICLE{Zepf:201936,
      author       = {Zepf, F. D. and Sungurtekin, I. and Glass, F. and Elstrodt,
                      L. and Peetz, D. and Hintereder, G. and Kratzsch, J. and
                      Biskup, C. S. and Poustka, F. and Wöckel, L.},
      title        = {{D}ifferences in zinc status and the leptin axis in
                      anorexic and recovered adolescents and young adults: a pilot
                      study},
      journal      = {Food $\&$ nutrition research},
      volume       = {56},
      number       = {0},
      issn         = {1654-661X},
      address      = {Häggeby [u.a.]},
      publisher    = {Co-Action Publishing},
      reportid     = {FZJ-2015-04226},
      pages        = {epub},
      year         = {2012},
      abstract     = {Background: Evidence from animal studies suggests that
                      leptin metabolism is associated with zinc (Zn) status.
                      However, research investigating this relationship in
                      adolescents and young adults with anorexia nervosa (AN) is
                      scarce; the present study aims to fill that gap.Methods:
                      Serum concentrations of leptin, the soluble leptin receptor
                      (sOB-R) and the free leptin index (FLI) were obtained in
                      healthy control subjects (n=19), acutely ill individuals
                      (n=14) and recovered patients with AN (n=15). Serum Zn
                      concentrations noted in previous research data were also
                      incorporated for all groups.Results: Leptin, FLI and Zn
                      concentrations were higher in recovered subjects with AN
                      when compared with acutely ill AN patients. Remitted
                      patients showed higher sOB-R concentrations but no
                      difference in FLI compared with the control group. Leptin
                      and FLI were lower in the acutely ill patients compared with
                      the control subjects, who showed no differences in Zn
                      concentrations. Zn concentrations were not correlated with
                      leptin, sOB-R or FLI concentrations in any of the three
                      investigated subgroups.Conclusions: The present
                      investigation does not entirely support an association
                      between Zn, Leptin and FLI concentrations in subjects with
                      AN, possibly due to limited statistical power. Further
                      research and replication of the present findings related to
                      the interaction between leptin and Zn is warranted. However,
                      with respect to serum leptin levels the data of the present
                      investigation indicate that acutely ill and remitted
                      patients with AN differ as regards serum leptin
                      concentrations and FLI, which is in line with previous
                      research.},
      cin          = {INM-3},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406},
      pnm          = {333 - Pathophysiological Mechanisms of Neurological and
                      Psychiatric Diseases (POF2-333)},
      pid          = {G:(DE-HGF)POF2-333},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000317117300001},
      pubmed       = {pmid:22393314},
      doi          = {10.3402/fnr.v56i0.10941},
      url          = {https://juser.fz-juelich.de/record/201936},
}