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@ARTICLE{Helmbold:201937,
author = {Helmbold, K. and Bubenzer, S. and Dahmen, B. and Eisert, A.
and Gaber, T. J. and Habel, U. and Konrad, K. and
Herpertz-Dahlmann, B. and Zepf, F. D.},
title = {{I}nfluence of acute tryptophan depletion on verbal
declarative episodic memory in young adult females},
journal = {Amino acids},
volume = {45},
number = {5},
issn = {1438-2199},
address = {Wien [u.a.]},
publisher = {Springer},
reportid = {FZJ-2015-04227},
pages = {1207 - 1219},
year = {2013},
abstract = {Diminished synthesis of the neurotransmitter serotonin
(5-HT) in the brain has been linked to disturbed memory
processes. The present study investigated the effects of
diminished central nervous 5-HT synthesis as achieved by an
acute dietary tryptophan depletion (ATD) on verbal
declarative episodic memory in young women while controlling
for the effects of female sex hormones. Eighteen healthy
females (aged 20–31 years) participated in a
within-subject repeated measures study, with two separate
days of assessment spaced at least one individual menstrual
cycle apart. On one day, participants were subjected to ATD,
thus lowering central nervous 5-HT synthesis. The other day
participants received a tryptophan-balanced amino acid load
(BAL = control condition). The study was randomized,
counterbalanced and double blind in terms of ATD/BAL
administration. Measurements took place in the early
follicular phase of the participants’ menstrual cycle.
Estrogen, FSH and LH levels were assessed at baseline.
Verbal declarative episodic memory was assessed using a
structured word-learning task. Short-term memory, as indexed
by immediate recall, was reduced after ATD intake, whereas
delayed recall and recognition after a 25-min delay did not
show any differences after intake of ATD or BAL. In young
women, verbal short-term memory function was more vulnerable
to ATD than consolidation processes. In light of the
possible interplay between female sex hormones and 5-HT,
further studies comparing different menstrual cycle phases
are needed.},
cin = {INM-3 / JARA-BRAIN},
ddc = {540},
cid = {I:(DE-Juel1)INM-3-20090406 / $I:(DE-82)080010_20140620$},
pnm = {333 - Pathophysiological Mechanisms of Neurological and
Psychiatric Diseases (POF2-333)},
pid = {G:(DE-HGF)POF2-333},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000325813800017},
pubmed = {pmid:24072504},
doi = {10.1007/s00726-013-1582-1},
url = {https://juser.fz-juelich.de/record/201937},
}