000202001 001__ 202001 000202001 005__ 20210129220003.0 000202001 0247_ $$2doi$$a10.1007/s00702-013-1119-5 000202001 0247_ $$2ISSN$$a0300-9564 000202001 0247_ $$2ISSN$$a1435-1463 000202001 0247_ $$2WOS$$aWOS:000333206100015 000202001 0247_ $$2altmetric$$aaltmetric:1954093 000202001 0247_ $$2pmid$$apmid:24292894 000202001 037__ $$aFZJ-2015-04290 000202001 082__ $$a610 000202001 1001_ $$0P:(DE-HGF)0$$avon Polier, G. G.$$b0 000202001 245__ $$aChange in electrodermal activity after acute tryptophan depletion associated with aggression in young people with attention deficit hyperactivity disorder (ADHD) 000202001 260__ $$aWien [u.a.]$$bSpringer$$c2014 000202001 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1435663714_1776 000202001 3367_ $$2DataCite$$aOutput Types/Journal article 000202001 3367_ $$00$$2EndNote$$aJournal Article 000202001 3367_ $$2BibTeX$$aARTICLE 000202001 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000202001 3367_ $$2DRIVER$$aarticle 000202001 4001_ $$aPolier, G. G. 000202001 520__ $$aWe investigated the impact of acute tryptophan depletion (ATD) and reduced brain serotonin synthesis on physiological arousal in 15 young people with ADHD participating in an aggression-inducing game. ATD was not associated with altered physiological arousal, as indexed by electrodermal activity (EDA). Baseline aggression was negatively correlated with the mean ATD effect on EDA. In accordance with the low arousal theory related to aggressive behavior, subjects with reduced physiological responsiveness/lower electrodermal reactivity to ATD tended to display elevated externalizing behavior. 000202001 536__ $$0G:(DE-HGF)POF2-333$$a333 - Pathophysiological Mechanisms of Neurological and Psychiatric Diseases (POF2-333)$$cPOF2-333$$fPOF II$$x0 000202001 588__ $$aDataset connected to CrossRef, juser.fz-juelich.de 000202001 7001_ $$0P:(DE-HGF)0$$aBiskup, C. S.$$b1 000202001 7001_ $$0P:(DE-HGF)0$$aKötting, W. F.$$b2 000202001 7001_ $$0P:(DE-Juel1)145464$$aBubenzer, S.$$b3 000202001 7001_ $$0P:(DE-HGF)0$$aHelmbold, K.$$b4 000202001 7001_ $$0P:(DE-HGF)0$$aEisert, A.$$b5 000202001 7001_ $$0P:(DE-HGF)0$$aGaber, T. J.$$b6 000202001 7001_ $$0P:(DE-HGF)0$$aZepf, F. D.$$b7$$eCorresponding Author 000202001 773__ $$0PERI:(DE-600)1481655-6$$a10.1007/s00702-013-1119-5$$gVol. 121, no. 4, p. 451 - 455$$n4$$p451 - 455$$tJournal of neural transmission$$v121$$x1435-1463$$y2014 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.pdf$$yRestricted 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.gif?subformat=icon$$xicon$$yRestricted 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.jpg?subformat=icon-1440$$xicon-1440$$yRestricted 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.jpg?subformat=icon-180$$xicon-180$$yRestricted 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.jpg?subformat=icon-640$$xicon-640$$yRestricted 000202001 8564_ $$uhttps://juser.fz-juelich.de/record/202001/files/art_10.1007_s00702-013-1119-5.pdf?subformat=pdfa$$xpdfa$$yRestricted 000202001 909CO $$ooai:juser.fz-juelich.de:202001$$pVDB 000202001 915__ $$0StatID:(DE-HGF)0100$$2StatID$$aJCR 000202001 915__ $$0StatID:(DE-HGF)0110$$2StatID$$aWoS$$bScience Citation Index 000202001 915__ $$0StatID:(DE-HGF)0111$$2StatID$$aWoS$$bScience Citation Index Expanded 000202001 915__ $$0StatID:(DE-HGF)0150$$2StatID$$aDBCoverage$$bWeb of Science Core Collection 000202001 915__ $$0StatID:(DE-HGF)0199$$2StatID$$aDBCoverage$$bThomson Reuters Master Journal List 000202001 915__ $$0StatID:(DE-HGF)0200$$2StatID$$aDBCoverage$$bSCOPUS 000202001 915__ $$0StatID:(DE-HGF)0300$$2StatID$$aDBCoverage$$bMedline 000202001 915__ $$0StatID:(DE-HGF)0310$$2StatID$$aDBCoverage$$bNCBI Molecular Biology Database 000202001 915__ $$0StatID:(DE-HGF)1030$$2StatID$$aDBCoverage$$bCurrent Contents - Life Sciences 000202001 915__ $$0StatID:(DE-HGF)1050$$2StatID$$aDBCoverage$$bBIOSIS Previews 000202001 915__ $$0StatID:(DE-HGF)9900$$2StatID$$aIF < 5 000202001 9101_ $$0I:(DE-588b)5008462-8$$6P:(DE-HGF)0$$aForschungszentrum Jülich GmbH$$b7$$kFZJ 000202001 9132_ $$0G:(DE-HGF)POF3-572$$1G:(DE-HGF)POF3-570$$2G:(DE-HGF)POF3-500$$aDE-HGF$$bKey Technologies$$lDecoding the Human Brain$$v(Dys-)function and Plasticity$$x0 000202001 9131_ $$0G:(DE-HGF)POF2-333$$1G:(DE-HGF)POF2-330$$2G:(DE-HGF)POF2-300$$3G:(DE-HGF)POF2$$4G:(DE-HGF)POF$$aDE-HGF$$bGesundheit$$lFunktion und Dysfunktion des Nervensystems$$vPathophysiological Mechanisms of Neurological and Psychiatric Diseases$$x0 000202001 9141_ $$y2015 000202001 920__ $$lyes 000202001 9201_ $$0I:(DE-Juel1)INM-3-20090406$$kINM-3$$lKognitive Neurowissenschaften$$x0 000202001 980__ $$ajournal 000202001 980__ $$aVDB 000202001 980__ $$aI:(DE-Juel1)INM-3-20090406 000202001 980__ $$aUNRESTRICTED