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@ARTICLE{Xu:202476,
      author       = {Xu, Changzheng and Tai, Huanhuan and Saleem, Muhammad and
                      Ludwig, Yvonne and Majer, Christine and Berendzen, Kenneth
                      W. and Nagel, Kerstin and Wojciechowski, Tobias and Meeley,
                      Robert B. and Taramino, Graziana and Hochholdinger, Frank},
      title        = {{C}ooperative action of the paralogous maize lateral organ
                      boundaries ({LOB}) domain proteins {RTCS} and {RTCL} in
                      shoot-borne root formation},
      journal      = {The new phytologist},
      volume       = {207},
      number       = {4},
      issn         = {0028-646X},
      address      = {Oxford [u.a.]},
      publisher    = {Wiley-Blackwell},
      reportid     = {FZJ-2015-04685},
      pages        = {1123 - 1133},
      year         = {2015},
      abstract     = {The paralogous maize (Zea mays) LBD (Lateral Organ
                      Boundaries Domain) genes rtcs (rootless concerning crown and
                      seminal roots) and rtcl (rtcs-like) emerged from an ancient
                      whole-genome duplication. RTCS is a key regulator of crown
                      root initiation. The diversity of expression, molecular
                      interaction and phenotype of rtcs and rtcl were
                      investigated. The rtcs and rtcl genes display highly
                      correlated spatio-temporal expression patterns in roots,
                      despite the significantly higher expression of rtcs. Both
                      RTCS and RTCL proteins bind to LBD downstream promoters and
                      act as transcription factors. In line with its auxin
                      inducibility and binding to auxin response elements of rtcs
                      and rtcl promoters, ARF34 (AUXIN RESPONSE FACTOR 34) acts as
                      transcriptional activator. Yeast two-hybrid screening
                      combined with bimolecular fluorescence complementation
                      (BiFC) experiments revealed conserved and unique interaction
                      partners of RTCS and RTCL. The rtcl mutation leads to
                      defective shoot-borne root elongation early in development.
                      Cooperative action of RTCS and RTCL during shoot-borne root
                      formation was demonstrated by rtcs-dependent repression of
                      rtcl transcription in coleoptilar nodes. Although RTCS is
                      instrumental in shoot-borne root initiation, RTCL controls
                      shoot-borne root elongation early in development. Their
                      conserved role in auxin signaling, but diverse function in
                      shoot-borne root formation, is underscored by their
                      conserved and unique interaction partners.},
      cin          = {IBG-2},
      ddc          = {580},
      cid          = {I:(DE-Juel1)IBG-2-20101118},
      pnm          = {582 - Plant Science (POF3-582)},
      pid          = {G:(DE-HGF)POF3-582},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000358965800021},
      pubmed       = {pmid:25902765},
      doi          = {10.1111/nph.13420},
      url          = {https://juser.fz-juelich.de/record/202476},
}