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@ARTICLE{Miotto:202935,
author = {Miotto, Marco C. and Valiente-Gabioud, Ariel A. and
Rossetti, Giulia and Zweckstetter, Markus and Carloni, Paolo
and Selenko, Philipp and Griesinger, Christian and Binolfi,
Andres and Fernández, Claudio O.},
title = {{C}opper {B}inding to the {N}-{T}erminally {A}cetylated,
{N}aturally {O}ccurring {F}orm of {A}lpha-{S}ynuclein
{I}nduces {L}ocal {H}elical {F}olding},
journal = {Journal of the American Chemical Society},
volume = {137},
number = {20},
issn = {1520-5126},
address = {Washington, DC},
publisher = {American Chemical Society},
reportid = {FZJ-2015-05061},
pages = {6444 - 6447},
year = {2015},
abstract = {Growing evidence supports a link between brain copper
homeostasis, the formation of alpha-synuclein (AS)-copper
complexes, and the development of Parkinson disease (PD).
Recently it was demonstrated that the physiological form of
AS is N-terminally acetylated (AcAS). Here we used NMR
spectroscopy to structurally characterize the interaction
between Cu(I) and AcAS. We found that the formation of an
AcAS–Cu(I) complex at the N-terminal region stabilizes
local conformations with α-helical secondary structure and
restricted motility. Our work provides new evidence into the
metallo-biology of PD and opens new lines of research as the
formation of AcAS–Cu(I) complex might impact on AcAS
membrane binding and aggregation.},
cin = {GRS / IAS-5 / JSC},
ddc = {540},
cid = {I:(DE-Juel1)GRS-20100316 / I:(DE-Juel1)IAS-5-20120330 /
I:(DE-Juel1)JSC-20090406},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511)},
pid = {G:(DE-HGF)POF3-511},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000355383500006},
pubmed = {pmid:25939020},
doi = {10.1021/jacs.5b01911},
url = {https://juser.fz-juelich.de/record/202935},
}