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@INPROCEEDINGS{Bos:256314,
author = {Bos, Hannah and Schücker, Jannis and Diesmann, Markus and
Helias, Moritz},
title = {{T}he anatomical origin of locally generated and induced
oscillations in a model of the cortical microcircuit},
reportid = {FZJ-2015-06277},
year = {2015},
abstract = {Fast oscillations of the population firing rate in the high
gamma range (50-200 Hz), as well as slow firing rate
fluctuations are ubiquitous in cortical recordings and have
been hypothesized to be generated locally []. Exploiting the
recently introduced multi-layered spiking neural network
model of a cortical microcircuit [2] as well as our
mean-field theoretical framework, we address the question of
the anatomical origin of the observed oscillations by
analyzing the spectra generated in the resting state
condition as well as under the application of constant and
oscillatory input.Deriving the theoretical framework we
perform a two-step reduction allowing for an incremental
validation of first the prediction of the population firing
rates and second the prediction of the population rate
spectra. Building on previous work deriving the mapping of
populations of leaky integrate-and-fire neurons to a linear
rate model [] and the response function of populations of
neurons connected by exponentially decaying synapses [1,3],
the mean-field framework is applicable to optional
circuitries set in the asynchronous irregular regime. In the
resting condition the neurons in the model fire irregularly,
displaying little synchrony on the population level.
Increasing the external input to the excitatory population
in layer 5 elicits slow rate fluctuations, reflected as
elevated slow frequency components in the population rate
spectra. Strengthening the input to the superficial layers
triggers population oscillations in the gamma range, while
the individual neurons preserve a firing pattern close to
irregularity with low rates. We derive a sensitivity measure
determining the anatomical connections within the circuit
crucial for the generation of the peaks visible in the power
spectra as well as their impact on and significance for the
frequencies and amplitudes. We identify a sub-circuit
located in layer 2/3 and 4 constituting the basis of the
high frequency oscillations, while connections within and
onto layer 5 determine the existence and strength of slow
rate fluctuations. Since the sensitivity measure is derived
from the mean-field theory we can analyze the robustness of
these findings under changes of parameters in the neuron and
synapse model and conclude on the actual contributions of
the anatomical connections given their embedding in the full
circuit.Exploiting the mean-field framework we generate
predictions regarding changes in the power spectra under
various stimulation protocols. Analyzing the responses of
the circuit to oscillatory input we formulate predictions
addressing the susceptibility of individual populations to
specific frequencies.},
month = {Oct},
date = {2015-10-17},
organization = {SfN, Chicago (USA), 17 Oct 2015 - 21
Oct 2015},
subtyp = {Outreach},
cin = {INM-6 / IAS-6},
cid = {I:(DE-Juel1)INM-6-20090406 / I:(DE-Juel1)IAS-6-20130828},
pnm = {571 - Connectivity and Activity (POF3-571) / 89571 -
Connectivity and Activity (POF2-89571) / HASB - Helmholtz
Alliance on Systems Biology (HGF-SystemsBiology) / MSNN -
Theory of multi-scale neuronal networks (HGF-SMHB-2014-2018)
/ SMHB - Supercomputing and Modelling for the Human Brain
(HGF-SMHB-2013-2017)},
pid = {G:(DE-HGF)POF3-571 / G:(DE-HGF)POF2-89571 /
G:(DE-Juel1)HGF-SystemsBiology /
G:(DE-Juel1)HGF-SMHB-2014-2018 /
G:(DE-Juel1)HGF-SMHB-2013-2017},
typ = {PUB:(DE-HGF)24},
url = {https://juser.fz-juelich.de/record/256314},
}