000279385 001__ 279385 000279385 005__ 20210129221045.0 000279385 0247_ $$2doi$$a10.1371/journal.pone.0118548 000279385 0247_ $$2Handle$$a2128/9571 000279385 0247_ $$2WOS$$aWOS:000350314700037 000279385 0247_ $$2altmetric$$aaltmetric:3750989 000279385 0247_ $$2pmid$$apmid:25734798 000279385 037__ $$aFZJ-2015-07397 000279385 082__ $$a500 000279385 1001_ $$0P:(DE-Juel1)144613$$aBorshchevskiy, Valentin$$b0 000279385 245__ $$aStructural and functional investigation of flavin binding center of the NqrC subunit of sodium-translocating NADH:Quinone oxidoreductase from Vibrio harveyi 000279385 260__ $$aLawrence, Kan.$$bPLoS$$c2015 000279385 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1449756504_5828 000279385 3367_ $$2DataCite$$aOutput Types/Journal article 000279385 3367_ $$00$$2EndNote$$aJournal Article 000279385 3367_ $$2BibTeX$$aARTICLE 000279385 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000279385 3367_ $$2DRIVER$$aarticle 000279385 520__ $$aNa+-translocating NADH:quinone oxidoreductase (NQR) is a redox-driven sodium pump operating in the respiratory chain of various bacteria, including pathogenic species. The enzyme has a unique set of redox active prosthetic groups, which includes two covalently bound flavin mononucleotide (FMN) residues attached to threonine residues in subunits NqrB and NqrC. The reason of FMN covalent bonding in the subunits has not been established yet. In the current work, binding of free FMN to the apo-form of NqrC from Vibrio harveyi was studied showing very low affinity of NqrC to FMN in the absence of its covalent bonding. To study structural aspects of flavin binding in NqrC, its holo-form was crystallized and its 3D structure was solved at 1.56 Å resolution. It was found that the isoalloxazine moiety of the FMN residue is buried in a hydrophobic cavity and that its pyrimidine ring is squeezed between hydrophobic amino acid residues while its benzene ring is extended from the protein surroundings. This structure of the flavin-binding pocket appears to provide flexibility of the benzene ring, which can help the FMN residue to take the bended conformation and thus to stabilize the one-electron reduced form of the prosthetic group. These properties may also lead to relatively weak noncovalent binding of the flavin. This fact along with periplasmic location of the FMN-binding domains in the vast majority of NqrC-like proteins may explain the necessity of the covalent bonding of this prosthetic group to prevent its loss to the external medium. 000279385 536__ $$0G:(DE-HGF)POF3-551$$a551 - Functional Macromolecules and Complexes (POF3-551)$$cPOF3-551$$fPOF III$$x0 000279385 588__ $$aDataset connected to CrossRef 000279385 7001_ $$0P:(DE-HGF)0$$aRound, Ekaterina$$b1 000279385 7001_ $$0P:(DE-HGF)0$$aBertsova, Yulia$$b2 000279385 7001_ $$0P:(DE-HGF)0$$aPolovinkin, Vitaly$$b3 000279385 7001_ $$0P:(DE-Juel1)165798$$aGushchin, Ivan$$b4$$ufzj 000279385 7001_ $$0P:(DE-Juel1)131968$$aIshchenko, Andrii$$b5 000279385 7001_ $$0P:(DE-Juel1)165629$$aKovalev, Kirill$$b6 000279385 7001_ $$0P:(DE-HGF)0$$aMishin, Alexey$$b7 000279385 7001_ $$0P:(DE-HGF)0$$aKachalova, Galina$$b8 000279385 7001_ $$0P:(DE-HGF)0$$aPopov, Alexander$$b9 000279385 7001_ $$0P:(DE-HGF)0$$aBogachev, Alexander$$b10 000279385 7001_ $$0P:(DE-Juel1)131964$$aGordeliy, Valentin$$b11$$eCorresponding author$$ufzj 000279385 773__ $$0PERI:(DE-600)2267670-3$$a10.1371/journal.pone.0118548$$gVol. 10, no. 3, p. e0118548 -$$n3$$pe0118548$$tPLoS one$$v10$$x1932-6203$$y2015 000279385 8564_ $$uhttps://juser.fz-juelich.de/record/279385/files/Structural%20and%20functional%20investigation%20of%20flavin%20binding%20center%20of%20the%20NqrC%20subunit%20of%20sodium-translocating%20NADH%3Aquinone%20oxidoreductase%20from%20Vibrio%20harveyi.pdf$$yOpenAccess 000279385 8564_ $$uhttps://juser.fz-juelich.de/record/279385/files/Structural%20and%20functional%20investigation%20of%20flavin%20binding%20center%20of%20the%20NqrC%20subunit%20of%20sodium-translocating%20NADH%3Aquinone%20oxidoreductase%20from%20Vibrio%20harveyi.gif?subformat=icon$$xicon$$yOpenAccess 000279385 8564_ 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