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@ARTICLE{BatraSafferling:49529,
author = {Batra-Safferling, R. and Abarca-Heidemann, K. and
Körschen, H. G. and Tziatzios, C. and Stoldt, M. and
Budyak, I. and Willbold, D. and Schwalbe, H. and
Klein-Seetharaman, J. and Kaupp, U. B.},
title = {{G}lutamic acid-rich proteins of rod photoreceptors are
natively unfolded},
journal = {The journal of biological chemistry},
volume = {281},
issn = {0021-9258},
address = {Bethesda, Md.},
publisher = {Soc.},
reportid = {PreJuSER-49529},
pages = {1449 - 1460},
year = {2006},
note = {Record converted from VDB: 12.11.2012},
abstract = {Broadly neutralizing HIV antibodies (bNAbs) can recognize
carbohydrate-dependent epitopes on gp120. In contrast to
previously characterized glycan-dependent bNAbs that
recognize high-mannose N-glycans, PGT121 binds complex-type
N-glycans in glycan microarrays. We isolated the B-cell
clone encoding PGT121, which segregates into PGT121-like and
10-1074-like groups distinguished by sequence, binding
affinity, carbohydrate recognition, and neutralizing
activity. Group 10-1074 exhibits remarkable potency and
breadth but no detectable binding to protein-free glycans.
Crystal structures of unliganded PGT121, 10-1074, and their
likely germ-line precursor reveal that differential
carbohydrate recognition maps to a cleft between
complementarity determining region (CDR)H2 and CDRH3. This
cleft was occupied by a complex-type N-glycan in a
"liganded" PGT121 structure. Swapping glycan contact
residues between PGT121 and 10-1074 confirmed their
importance for neutralization. Although PGT121 binds
complex-type N-glycans, PGT121 recognized high-mannose-only
HIV envelopes in isolation and on virions. As HIV envelopes
exhibit varying proportions of high-mannose- and
complex-type N-glycans, these results suggest promiscuous
carbohydrate interactions, an advantageous adaptation
ensuring neutralization of all viruses within a given
strain.},
keywords = {J (WoSType)},
cin = {IBI-1 / IBI-2},
ddc = {570},
cid = {I:(DE-Juel1)VDB57 / I:(DE-Juel1)VDB58},
pnm = {Funktion und Dysfunktion des Nervensystems},
pid = {G:(DE-Juel1)FUEK409},
shelfmark = {Biochemistry $\&$ Molecular Biology},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:23115339},
UT = {WOS:000234652000022},
doi = {10.1074/jbcM505012200},
url = {https://juser.fz-juelich.de/record/49529},
}