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@ARTICLE{Moukhametzianov:50688,
author = {Moukhametzianov, R. and Klare, J. P. and Efremov, R. and
Baeken, C. and Göppner, A. and Labahn, J. and Engelhard, M.
and Büldt, G. and Gordeliy, V. I.},
title = {{D}evelopment of the signal in sensory rhodopsin and its
transfer to the cognate transducer},
journal = {Nature},
volume = {440},
issn = {0028-0836},
address = {London [u.a.]},
publisher = {Nature Publising Group},
reportid = {PreJuSER-50688},
pages = {115 - 119},
year = {2006},
note = {Record converted from VDB: 12.11.2012},
abstract = {The microbial phototaxis receptor sensory rhodopsin II
(NpSRII, also named phoborhodopsin) mediates the photophobic
response of the haloarchaeon Natronomonas pharaonis by
modulating the swimming behaviour of the bacterium. After
excitation by blue-green light NpSRII triggers, by means of
a tightly bound transducer protein (NpHtrII), a signal
transduction chain homologous with the two-component system
of eubacterial chemotaxis. Two molecules of NpSRII and two
molecules of NpHtrII form a 2:2 complex in membranes as
shown by electron paramagnetic resonance and X-ray structure
analysis. Here we present X-ray structures of the photocycle
intermediates K and late M (M2) explaining the evolution of
the signal in the receptor after retinal isomerization and
the transfer of the signal to the transducer in the complex.
The formation of late M has been correlated with the
formation of the signalling state. The observed structural
rearrangements allow us to propose the following mechanism
for the light-induced activation of the signalling complex.
On excitation by light, retinal isomerization leads in the K
state to a rearrangement of a water cluster that partly
disconnects two helices of the receptor. In the transition
to late M the changes in the hydrogen bond network proceed
further. Thus, in late M state an altered tertiary structure
establishes the signalling state of the receptor. The
transducer responds to the activation of the receptor by a
clockwise rotation of about 15 degrees of helix TM2 and a
displacement of this helix by 0.9 A at the cytoplasmic
surface.},
keywords = {Biological Evolution / Crystallography, X-Ray / Cytoplasm:
metabolism / Halobacteriaceae: chemistry / Halobacteriaceae:
cytology / Halobacteriaceae: metabolism / Halorhodopsins:
chemistry / Halorhodopsins: metabolism / Hydrogen Bonding /
Isomerism / Light Signal Transduction: physiology / Models,
Molecular / Protein Conformation / Sensory Rhodopsins:
chemistry / Sensory Rhodopsins: metabolism / Halorhodopsins
(NLM Chemicals) / Sensory Rhodopsins (NLM Chemicals) / J
(WoSType)},
cin = {IBI-2},
ddc = {070},
cid = {I:(DE-Juel1)VDB58},
pnm = {Funktion und Dysfunktion des Nervensystems},
pid = {G:(DE-Juel1)FUEK409},
shelfmark = {Multidisciplinary Sciences},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:16452929},
UT = {WOS:000235685700051},
doi = {10.1038/nature04520},
url = {https://juser.fz-juelich.de/record/50688},
}