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@ARTICLE{Mller:5449,
author = {Müller, J. and Reyes-Haro, D. and Rivneva, T. and Nolte,
C. and Schaette, R. and Lübke, J.H.R. and Kettenmann, H.},
title = {{T}he principal neurons of the medial nucleus of the
trapezoid body and {NG}2+ glial cells receive coordinated
excitatory synaptic input.},
journal = {The journal of general physiology},
volume = {134},
issn = {0022-1295},
address = {New York, NY},
publisher = {Rockefeller Univ. Press},
reportid = {PreJuSER-5449},
pages = {115 - 127},
year = {2009},
note = {We thank R. Juttner for discussions and comments on the
manuscript and for sharing chemicals, K. Karram and J.
Trotter for providing antibodies, and R. Krober for
technical assistance.This study was supported by a grant
from the Deutsche Forschungsgemeinschaft (SPP 1172).},
abstract = {Glial cell processes are part of the synaptic structure and
sense spillover of transmitter, while some glial cells can
even receive direct synaptic input. Here, we report that a
defined type of glial cell in the medial nucleus of the
trapezoid body (MNTB) receives excitatory glutamatergic
synaptic input from the calyx of Held (CoH). This giant
glutamatergic terminal forms an axosomatic synapse with a
single principal neuron located in the MNTB. The NG2 glia,
as postsynaptic principal neurons, establish synapse-like
structures with the CoH terminal. In contrast to the
principal neurons, which are known to receive excitatory as
well as inhibitory inputs, the NG2 glia receive mostly, if
not exclusively,
alpha-amino-3-hydroxy-5-methyl-isoxazole-4-propionic acid
receptor-mediated evoked and spontaneous synaptic input.
Simultaneous recordings from neurons and NG2 glia indicate
that they partially receive synchronized spontaneous input.
This shows that an NG2(+) glial cell and a postsynaptic
neuron share presynaptic terminals.},
keywords = {Animals / Astrocytes: metabolism / Auditory Pathways:
physiology / Brain Stem: cytology / Brain Stem: physiology /
Green Fluorescent Proteins: genetics / Green Fluorescent
Proteins: metabolism / Immunohistochemistry / Mice / Mice,
Transgenic / Microscopy, Electron / Neuroglia: metabolism /
Neurons: metabolism / Receptors, AMPA: metabolism / Signal
Transduction / Synaptic Transmission: physiology /
Receptors, AMPA (NLM Chemicals) / Green Fluorescent Proteins
(NLM Chemicals) / J (WoSType)},
cin = {INM-2 / JARA-BRAIN},
ddc = {610},
cid = {I:(DE-Juel1)INM-2-20090406 / $I:(DE-82)080010_20140620$},
pnm = {Funktion und Dysfunktion des Nervensystems},
pid = {G:(DE-Juel1)FUEK409},
shelfmark = {Physiology},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:19635853},
pmc = {pmc:PMC2717692},
UT = {WOS:000268367600003},
doi = {10.1085/jgp.200910194},
url = {https://juser.fz-juelich.de/record/5449},
}