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@ARTICLE{Vossel:807876,
author = {Vossel, Simone and Fink, Gereon Rudolf},
title = {{C}ontralesional distractors enhance ipsilesional target
processing after righthemispheric stroke},
journal = {Cortex},
volume = {78},
issn = {0010-9452},
address = {Paris},
publisher = {Elsevier Masson},
reportid = {FZJ-2016-02204},
pages = {115-124},
year = {2016},
abstract = {Stroke can result in marked impairments in the processing
of information presented in contralesional space. The
present prospective study investigated how a contralesional
distractor affects ipsilesional perception in patients with
a right-hemispheric stroke. In a simple target detection
task, the influence of the distractor on reaction times
(RTs) was examined in stroke patients and compared to the
performance of healthy elderly controls. Distractor
interference effects were related to measures of neglect and
extinction using a regression analysis. Moreover, the
magnitude of the behavioural distractor effect entered a
voxel-based lesion-symptom mapping (VLSM) analysis with the
hypothesis that parietal and temporoparietal lesions are
related to altered distractor processing. While the presence
of a distractor in the opposite hemifield slowed down RTs in
healthy controls for left and right targets, this effect was
only observed for contralesional left targets in the group
of right-hemispheric patients. In stark contrast, the
presence of a distractor in the contralesional hemifield
expedited ipsilesional (i.e., right) target detection. This
effect was significantly related to lesions in the anterior
middle temporal and temporoparietal cortex, external and
internal capsule, as well as the superior longitudinal
fascicle (SLF). These results elucidate the nature of the
disruption of attentive processing in the contralesional
hemifield after right-hemispheric stroke. More specifically,
they shed light on the abnormal prioritisation of
ipsilesional information: our data suggest that damage to
the temporal and temporoparietal cortex and white matter
tracts may transform contralesional stimulation into an
unspecific saliency signal contributing to facilitated
information processing in ipsilesional space.},
cin = {INM-3},
ddc = {570},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000376715000009},
pubmed = {pmid:27035700},
doi = {10.1016/j.cortex.2016.02.010},
url = {https://juser.fz-juelich.de/record/807876},
}