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@ARTICLE{Dillen:810729,
      author       = {Dillen, Kim N. H. and Jacobs, Heidi I. L. and Kukolja,
                      Juraj and von Reutern, Boris and Richter, Nils and Onur,
                      Özgür A. and Dronse, Julian and Langen, Karl-Josef and
                      Fink, Gereon R.},
      title        = {{A}berrant functional connectivity differentiates
                      retrosplenial cortex from posterior cingulate cortex in
                      prodromal {A}lzheimer's disease},
      journal      = {Neurobiology of aging},
      volume       = {44},
      issn         = {0197-4580},
      address      = {Amsterdam [u.a.]},
      publisher    = {Elsevier Science},
      reportid     = {FZJ-2016-03321},
      pages        = {114 - 126},
      year         = {2016},
      abstract     = {The posterior cingulate cortex (PCC) is a key hub of the
                      default mode network, a resting-state network involved in
                      episodic memory, showing functional connectivity (FC)
                      changes in Alzheimer's disease (AD). However, PCC is a
                      cytoarchitectonically heterogeneous region. Specifically,
                      the retrosplenial cortex (RSC), often subsumed under the
                      PCC, is an area functionally and microanatomically distinct
                      from PCC. To investigate FC patterns of RSC and PCC
                      separately, we used resting-state functional magnetic
                      resonance imaging in healthy aging participants, patients
                      with subjective cognitive impairment, and prodromal AD.
                      Compared to the other 2 groups, we found higher FC from RSC
                      to frontal cortex in subjective cognitive impairment but
                      higher FC to occipital cortex in prodromal AD. Conversely,
                      FC from PCC to the lingual gyrus was higher in prodromal AD.
                      Furthermore, data indicate that RSC and PCC are
                      characterized by differential FC patterns represented by
                      hub-specific interactions with memory and attentions scores
                      in prodromal AD compared to cognitively normal individuals,
                      possibly reflecting compensatory mechanisms for RSC and
                      neurodegenerative processes for PCC. Data thus confirm and
                      extend previous studies suggesting that the RSC is
                      functionally distinct from PCC.},
      cin          = {INM-3 / INM-4},
      ddc          = {610},
      cid          = {I:(DE-Juel1)INM-3-20090406 / I:(DE-Juel1)INM-4-20090406},
      pnm          = {572 - (Dys-)function and Plasticity (POF3-572)},
      pid          = {G:(DE-HGF)POF3-572},
      typ          = {PUB:(DE-HGF)16},
      UT           = {WOS:000378058500011},
      pubmed       = {pmid:27318139},
      doi          = {10.1016/j.neurobiolaging.2016.04.010},
      url          = {https://juser.fz-juelich.de/record/810729},
}