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@ARTICLE{Rabenstein:820103,
author = {Rabenstein, Monika and Vay, Sabine Ulrike and Flitsch, Lea
Jessica and Fink, Gereon Rudolf and Schroeter, Michael and
Rueger, Maria Adele},
title = {{O}steopontin directly modulates cytokine expression of
primary microglia and increases their survival},
journal = {Journal of neuroimmunology},
volume = {299},
issn = {0165-5728},
address = {Amsterdam [u.a.]},
publisher = {Elsevier Science},
reportid = {FZJ-2016-05654},
pages = {130 - 138},
year = {2016},
abstract = {Osteopontin (OPN) is constitutively expressed in the brain
and upregulated during neuroinflammation, e.g., focal
cerebral ischemia. In OPN-deficient mice, microglia are
deregulated after ischemia, but specific OPN-effects on
microglia remain elusive.Primary microglia were cultured in
the presence or absence of OPN. The survival of microglia
under stress conditions was dose-dependently increased by
OPN. Lipopolysaccharides (LPS)-induced release of nitric
oxide (NO), TNF-α, and IL-6, as well as expression of
inducible Nitric Oxide Synthase (iNOS), were attenuated by
OPN. Data suggest that OPN modulates microglia function by
shifting their inflammatory profile towards a neutral
anti-inflammatory phenotype.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000386190900021},
pubmed = {pmid:27725111},
doi = {10.1016/j.jneuroim.2016.09.009},
url = {https://juser.fz-juelich.de/record/820103},
}