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@ARTICLE{LaSala:820730,
author = {La Sala, Giuseppina and Riccardi, Laura and Gaspari,
Roberto and Cavalli, Andrea and Hantschel, Oliver and De
Vivo, Marco},
title = {{HRD} {M}otif as the {C}entral {H}ub of the {S}ignaling
{N}etwork for {A}ctivation {L}oop {A}utophosphorylation in
{A}bl {K}inase},
journal = {Journal of chemical theory and computation},
volume = {12},
number = {11},
issn = {1549-9626},
address = {Washington, DC},
reportid = {FZJ-2016-05998},
pages = {5563 - 5574},
year = {2016},
abstract = {A number of structural factors modulate the activity of
Abelson (Abl) tyrosine kinase, whose deregulation is often
related to oncogenic processes. First, only the open
conformation of the Abl kinase domain’s activation loop
(A-loop) favors ATP binding to the catalytic cleft. In this
regard, the trans-autophosphorylation of the Y412 residue,
which is located along the A-loop, favors the stability of
the open conformation, in turn enhancing Abl activity.
Another key factor for full Abl activity is the formation of
active conformations of the catalytic DFG motif in the Abl
kinase domain. Furthermore, binding of the SH2 domain to the
N-lobe of the Abl kinase was recently demonstrated to have a
long-range allosteric effect on the stabilization of the
A-loop open state. Intriguingly, these distinct structural
factors imply a complex signal transmission network for
controlling the A-loop’s flexibility and conformational
preference for optimal Abl function. However, the exact
dynamical features of this signal transmission network
structure remain unclear. Here, we report on
microsecond-long molecular dynamics coupled with enhanced
sampling simulations of multiple Abl model systems, in the
presence or absence of the SH2 domain and with the DFG motif
flipped in two ways (in or out conformation). Through
comparative analysis, our simulations augment the
interpretation of the existing Abl experimental data,
revealing a dynamical network of interactions that
interconnect SH2 domain binding with A-loop plasticity and
Y412 autophosphorylation in Abl. This signaling network
engages the DFG motif and, importantly, other conserved
structural elements of the kinase domain, namely, the
EPK-ELK H-bond network and the HRD motif. Our results show
that the signal propagation for modulating the A-loop
spatial localization is highly dependent on the HRD motif
conformation, which thus acts as the central hub of this
(allosteric) signaling network controlling Abl activation
and function.},
cin = {IAS-5 / INM-9},
ddc = {540},
cid = {I:(DE-Juel1)IAS-5-20120330 / I:(DE-Juel1)INM-9-20140121},
pnm = {899 - ohne Topic (POF3-899)},
pid = {G:(DE-HGF)POF3-899},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000387519400031},
pubmed = {pmid:27682200},
doi = {10.1021/acs.jctc.6b00600},
url = {https://juser.fz-juelich.de/record/820730},
}