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@ARTICLE{Bischof:825911,
author = {Bischof, Gérard N. and Jessen, Frank and Fliessbach, Klaus
and Dronse, Julian and Hammes, Jochen and Neumaier, Bernd
and Onur, Oezguer and Fink, Gereon R. and Kukolja, Juraj and
Drzezga, Alexander and van Eimeren, Thilo},
title = {{I}mpact of tau and amyloid burden on glucose metabolism in
{A}lzheimer's disease.},
journal = {Annals of Clinical and Translational Neurology},
volume = {3},
number = {12},
issn = {2328-9503},
address = {Chichester [u.a.]},
publisher = {Wiley},
reportid = {FZJ-2017-00190},
pages = {934 - 939},
year = {2016},
abstract = {In a multimodal PET imaging approach, we determined the
differential contribution of neurofibrillary tangles
(measured with [18F]AV-1451) and beta-amyloid burden
(measured with [11C]PiB) on degree of neurodegeneration
(i.e., glucose metabolism measured with [18F]FDG-PET) in
patients with Alzheimer's disease. Across brain regions, we
observed an interactive effect of beta-amyloid burden and
tau deposition on glucose metabolism which was most
pronounced in the parietal lobe. Elevated beta-amyloid
burden was associated with a stronger influence of tau
accumulation on glucose metabolism. Our data provide the
first in vivo insights into the differential contribution of
Aβ and tau to neurodegeneration in Alzheimer's disease.},
cin = {INM-3 / INM-5},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406 / I:(DE-Juel1)INM-5-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000390566700004},
pubmed = {pmid:28097205},
doi = {10.1002/acn3.339},
url = {https://juser.fz-juelich.de/record/825911},
}