% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Oberwelland:827727,
author = {Oberwelland, E. and Schilbach, L. and Barisic, I. and
Krall, S. C. and Vogeley, K. and Fink, G. R. and
Herpertz-Dahlmann, B. and Konrad, K. and Schulte-Rüther,
M.},
title = {{Y}oung adolescents with autism show abnormal joint
attention network: {A} gaze contingent f{MRI} study},
journal = {NeuroImage: Clinical},
volume = {14},
issn = {2213-1582},
address = {[Amsterdam u.a.]},
publisher = {Elsevier},
reportid = {FZJ-2017-01835},
pages = {112 - 121},
year = {2017},
abstract = {Behavioral research has revealed deficits in the
development of joint attention (JA) as one of the earliest
signs of autism. While the neural basis of JA has been
studied predominantly in adults, we recently demonstrated a
protracted development of the brain networks supporting JA
in typically developing children and adolescents. The
present eye-tracking/fMRI study now extends these findings
to adolescents with autism. Our results show that in
adolescents with autism JA is subserved by abnormal
activation patterns in brain areas related to social
cognition abnormalities which are at the core of ASD
including the STS and TPJ, despite behavioral maturation
with no behavioral differences. Furthermore, in the autism
group we observed increased neural activity in a network of
social and emotional processing areas during interactions
with their mother. Moreover, data indicated that less
severely affected individuals with autism showed higher
frontal activation associated with self-initiated
interactions. Taken together, this study provides first-time
data of JA in children/adolescents with autism incorporating
the interactive character of JA, its reciprocity and
motivational aspects. The observed functional differences in
adolescents ASD suggest that persistent developmental
differences in the neural processes underlying JA contribute
to social interaction difficulties in ASD.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000405984300012},
pubmed = {pmid:28180069},
doi = {10.1016/j.nicl.2017.01.006},
url = {https://juser.fz-juelich.de/record/827727},
}