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@ARTICLE{Li:827728,
author = {Li, Jinyu and Vervoorts, Jörg and Carloni, Paolo and
Rossetti, Giulia and Lüscher, Bernhard},
title = {{S}tructural prediction of the interaction of the tumor
suppressor p27$^{{KIP}1}$ with cyclin {A}/{CDK}2 identifies
a novel catalytically relevant determinant},
journal = {BMC bioinformatics},
volume = {18},
number = {1},
issn = {1471-2105},
address = {London},
publisher = {BioMed Central},
reportid = {FZJ-2017-01836},
pages = {15},
year = {2017},
abstract = {Background The cyclin-dependent kinase 2 (CDK2) together
with its cyclin E and A partners is a central regulator of
cell growth and division. Deregulation of CDK2 activity is
associated with diseases such as cancer. The analysis of
substrates identified S/T-P-X-R/K/H as the CDK2 consensus
sequence. The crystal structure of cyclin A/CDK2 with a
short model peptide supports this sequence and identifies
key interactions. However, CDKs use additional determinants
to recognize substrates, including the RXL motif that is
read by the cyclin subunits. We were interested to determine
whether additional amino acids beyond the minimal consensus
sequence of the well-studied substrate and tumor suppressor
p27KIP1 were relevant for catalysis.ResultsTo address
whether additional amino acids, close to the minimal
consensus sequence, play a role in binding, we investigate
the interaction of cyclin A/CDK2 with an in vivo cellular
partner and CDK inhibitor p27KIP1. This protein is an
intrinsically unfolded protein and, in particular, the
C-terminal half of the protein has not been accessible to
structural analysis. This part harbors the CDK2
phosphorylation site. We used bioinformatics tools,
including MODELLER, iTASSER and HADDOCK, along with partial
structural information to build a model of the C-terminal
region of p27KIP1 with cyclin A/CDK2. This revealed novel
interactions beyond the consensus sequence with a proline
and a basic amino acid at the P + 1 and the P + 3
sites, respectively. We suggest that the lysine at P + 2
might regulate the reversible association of the second
counter ion in the active site of CDK2. The arginine at
P + 7 interacts with both cyclin A and CDK2 and is
important for the catalytic turnover rate.ConclusionOur
modeling identifies additional amino acids in p27KIP1 beyond
the consensus sequence that contribute to the efficiency of
substrate phosphorylation.},
cin = {IAS-5 / INM-9 / JSC},
ddc = {004},
cid = {I:(DE-Juel1)IAS-5-20120330 / I:(DE-Juel1)INM-9-20140121 /
I:(DE-Juel1)JSC-20090406},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511)},
pid = {G:(DE-HGF)POF3-511},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000392170400003},
pubmed = {pmid:28056778},
doi = {10.1186/s12859-016-1411-0},
url = {https://juser.fz-juelich.de/record/827728},
}