000830498 001__ 830498 000830498 005__ 20240625095111.0 000830498 0247_ $$2doi$$a10.1016/j.bmcl.2017.04.026 000830498 0247_ $$2ISSN$$a0960-894X 000830498 0247_ $$2ISSN$$a1464-3405 000830498 0247_ $$2WOS$$aWOS:000402472600004 000830498 037__ $$aFZJ-2017-04039 000830498 082__ $$a540 000830498 1001_ $$0P:(DE-HGF)0$$aHendriks, Christine M. M.$$b0 000830498 245__ $$aSulfoximines as ATR inhibitors: Analogs of VE-821 000830498 260__ $$aAmsterdam [u.a.]$$bElsevier Science$$c2017 000830498 3367_ $$2DRIVER$$aarticle 000830498 3367_ $$2DataCite$$aOutput Types/Journal article 000830498 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1497010275_12002 000830498 3367_ $$2BibTeX$$aARTICLE 000830498 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000830498 3367_ $$00$$2EndNote$$aJournal Article 000830498 520__ $$aThe ATM- and Rad3-related (ATR) kinases play a key role in DNA repair processes and thus ATR is an attractive target for cancer therapy. Here we designed and synthesized sulfilimidoyl- and sulfoximidoyl-substituted analogs of the sulfone VE-821, a reported ATR inhibitor. The properties of these analogs have been investigated by calculating physicochemical parameters and studying their potential to specifically inhibit ATR in cells. Prolonged inhibition of ATR by the analogs in a Burkitt lymphoma cell line resulted in enhanced DNA damage and a substantial amount of apoptosis. 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