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@ARTICLE{Schneider:835112,
author = {Schneider, Christian and Wunderlich, Gilbert and Bleistein,
Johannes and Fink, Gereon Rudolf and Deckert, Martina and
Brunn, Anna and Lehmann, Helmar Christoph},
title = {{L}ymphocyte antigens targetable by monoclonal antibodies
in non-systemic vasculitic neuropathy},
journal = {Journal of neurology, neurosurgery, and psychiatry},
volume = {88},
number = {9},
issn = {1468-330X},
address = {London},
publisher = {BMJ Publishing Group},
reportid = {FZJ-2017-04980},
pages = {756–760},
year = {2017},
abstract = {Objective To identify the most relevant antigens for
monoclonal antibodies in lymphocytic infiltrates in
non-systemic vasculitic neuropathy (NSVN).Background Current
immunosuppressive treatment for NSVN is insufficient.
Monoclonal antibodies might be a treatment option, but the
expression profile for targetable antigens on lymphocytic
infiltrates in NSVN is unknown.Methods Sural nerve biopsies
from a cohort of patients with NSVN were
immunohistochemically studied for the expression of
potential candidate antigens in perivascular and intramural
lymphocytic infiltrates and correlated with neurological and
electrophysiological parameters. 20 patients with
treatment naïve NSVN and 5 patients with idiopathic axonal
neuropathy were included.Results The CD52, BAFF and CD49d
antigens were expressed in epineurial, perivascular or
intramural lymphocytes of all (20/20) patients. CD52 was
most prominently expressed in $21.49\%$ of all inflammatory
infiltrates. BAFF and CD49d were detected in $11.25\%$ and
$10.99\%$ of these lymphocytes, respectively. The CD20, CD25
and CD126 antigens were found less frequently and at low
levels only (CD20: 10/20 patients, $5.84\%$ of lymphocytes;
CD25: 17/20 patients, $5.22\%$ of lymphocytes; CD126: 3/20
patients, $0.15\%$ of lymphocytes).Conclusion This is the
first study in NSVN that identifies antigens expressed by
pathogenic lymphocytes, which are potential targets for
future monoclonal antibody treatment. Our data suggest that
NSVN is amenable to monoclonal antibodies and, moreover,
that targeting CD52 may be particularly promising. Our
results strongly warrant future clinical trials in NSVN with
monoclonal antibodies.},
cin = {INM-3},
ddc = {610},
cid = {I:(DE-Juel1)INM-3-20090406},
pnm = {572 - (Dys-)function and Plasticity (POF3-572)},
pid = {G:(DE-HGF)POF3-572},
typ = {PUB:(DE-HGF)16},
UT = {WOS:000407777300010},
pubmed = {pmid:28550073},
doi = {10.1136/jnnp-2017-315878},
url = {https://juser.fz-juelich.de/record/835112},
}