Hauptseite > Publikationsdatenbank > Investigation of common, low-frequency and rare genome-wide variation in anorexia nervosa. > print

001     841233
005     20210129231941.0
024 7 _ |a 10.1038/mp.2017.88
|2 doi
024 7 _ |a pmid:29155802
|2 pmid
024 7 _ |a 1359-4184
|2 ISSN
024 7 _ |a 1476-5578
|2 ISSN
024 7 _ |a 2128/18482
|2 Handle
024 7 _ |a WOS:000431614600009
|2 WOS
024 7 _ |a altmetric:22389816
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037 _ _ |a FZJ-2017-08325
041 _ _ |a eng
082 _ _ |a 610
100 1 _ |a Huckins, L. M.
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245 _ _ |a Investigation of common, low-frequency and rare genome-wide variation in anorexia nervosa.
260 _ _ |a London
|c 2018
|b Macmillan
336 7 _ |a article
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336 7 _ |a Output Types/Journal article
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336 7 _ |a Journal Article
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336 7 _ |a ARTICLE
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336 7 _ |a JOURNAL_ARTICLE
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336 7 _ |a Journal Article
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520 _ _ |a Anorexia nervosa (AN) is a complex neuropsychiatric disorder presenting with dangerously low body weight, and a deep and persistent fear of gaining weight. To date, only one genome-wide significant locus associated with AN has been identified. We performed an exome-chip based genome-wide association studies (GWAS) in 2158 cases from nine populations of European origin and 15 485 ancestrally matched controls. Unlike previous studies, this GWAS also probed association in low-frequency and rare variants. Sixteen independent variants were taken forward for in silico and de novo replication (11 common and 5 rare). No findings reached genome-wide significance. Two notable common variants were identified: rs10791286, an intronic variant in OPCML (P=9.89 × 10-6), and rs7700147, an intergenic variant (P=2.93 × 10-5). No low-frequency variant associations were identified at genome-wide significance, although the study was well-powered to detect low-frequency variants with large effect sizes, suggesting that there may be no AN loci in this genomic search space with large effect sizes.Molecular Psychiatry advance online publication, 25 July 2017; doi:10.1038/mp.2017.88.
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