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@INPROCEEDINGS{Schmitz:842638,
author = {Schmitz, Sabine and Pinkawa, Michael and Eble, Michael and
Kriehuber, Ralf},
title = {{P}ersisting ring chromosomes detected by m{FISH} in
{L}ymphocytes of a cancer patient - a case study},
reportid = {FZJ-2018-00845},
year = {2013},
abstract = {Background: We report the case of an 84 years old prostate
cancer patient with severe side effects after radiotherapy
in 2006. He was cytogenetically analysed in 2009 and in 2012
in a comparative study for individual radiosensitivity of
prostate cancer patients. No other patient had clonal
aberrations, but this patient showed ring chromosomes in the
range of 21-25 $\%$ of lymphocytes. He received 5 cycles of
5-fluorouracil/folic acid for chemotherapy of sigmoid colon
carcinoma in 2003, three years before radiotherapy of
prostate cancer.Material und Methods: Blood samples were
irradiated ex vivo with Cs-137 γ-rays (0.7 Gy/min) in the
G0-phase of the cell cycle. 100 FISH painted metaphases were
analysed for the control and the irradiated samples each.
Multicolour in situ hybridisation techniques like mFISH and
mBand as well as MYC locus, telomere and centromere painting
probes were used to characterize ring metaphases. Metaphase
search and autocapture was performed with a Zeiss Axioplan 2
imaging microscope followed by scoring and image analysis
using Metafer 4/ISIS software (MetaSystems).Results: In 2009
chromosome 8 rings were found in about 25 $\%$ of
lymphocytes. Rings were stable over time and increased to
about 30 $\%$ until 2012. The ring chro-mosome 8 always
lacked telomere signals and a small amount of rings
displayed up to four centromere signals. In aberrant
metaphases 8pter and 8qter were either translocated or
deleted. Further analyses revealed that the breakpoint at
the p arm is localized at 8p21.2-22. The breakpoint at the q
arm turned out to be distal from the MYC locus at
8q23-24.Conclusion: We hypothesize that the ring chromosome
8 has been developed dur-ing the 5 FU/folic acid treatments
in 2003. The long term persistence might be due to clonal
expansion of a damaged but viable hematopoietic stem cell
giving rise to cy-cling progenitor cells that permit cell
survival and proliferation.Funded by Dr.
Erich-Schmitt-Stiftung},
month = {Sep},
date = {2013-09-25},
organization = {16th Annual Meeting of the German
Society for Biological Radiation
Research, Darmstadt (Germay), 25 Sep
2013 - 27 Sep 2013},
subtyp = {Invited},
cin = {S-US},
cid = {I:(DE-Juel1)S-US-20090406},
pnm = {899 - ohne Topic (POF3-899)},
pid = {G:(DE-HGF)POF3-899},
typ = {PUB:(DE-HGF)6},
url = {https://juser.fz-juelich.de/record/842638},
}