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@INPROCEEDINGS{Gertzen:844071,
author = {Gertzen, C. G. W. and Keitel, V. and Seidel, C. A. M. and
Gohlke, H.},
title = {{U}nravelling the {D}i- and {O}ligomerisation {I}nterfaces
of the {G}-{P}rotein {C}oupled {B}ile {A}cid {R}eceptor
{TGR}5 via {I}ntegrative {M}odelling},
volume = {49},
address = {Jülich},
publisher = {John von Neumann Institute for Computing},
reportid = {FZJ-2018-01580},
series = {Publication Series of the John von Neumann Institute for
Computing (NIC) NIC Series},
pages = {25 - 31},
year = {2018},
abstract = {TGR5 is a bile acid- and neurosteroid-sensing G-protein
coupled receptor (GPCR), which isalmost ubiquitously
expressed throughout the human body. Its physiological
functions comprisethe regulation of blood glucose
homeostasis, metabolism, and inflammation.
Additionally,recent studies show an involvement of TGR5 in
the formation of gastric, esophageal, andcholangiocyte
cancers as well as in bile acid-induced itch. Hence, TGR5
has been identified asan important drug target. To reduce
side effects of drugs targeting GPCRs, the development
ofbivalent ligands specifically targeting dimers was shown
to be promising. To do so, the knowledgeof the dimerisation
interfaces of these GPCRs is paramount. However, the
dimerisationinterfaces of TGR5 are not known. Here, we
present the identification of the primary
dimerisationinterface of TGR5 and possible oligomerisation
interfaces. We used Multiparameter ImageFluorescence
Spectroscopy (MFIS) Förster Resonance Energy Transfer
(FRET) measurementsof fluorescently labelled TGR5 in live
cells to measure apparent distances between two
TGR5protomers and compared them to distances computed for
putative TGR5 dimer models. Asthe linker between TGR5 and
the fluorophores contained more than 30 residues, we used
all-atommolecular dynamics (MD) simulations to sample the
conformational space of the linkerand fluorophore in
relation to TGR5. The sampled configurations were reweighted
by free energycalculations using the molecular mechanics
Poisson-Boltzmann surface area (MM-PBSA)method to account
for the presence of solvent and a membrane, and a random
energy model toestimate the configurational entropy. This
allowed us to identify the 1-8 interface of TGR5 asthe
primary dimerisation interface, with the 4-5 and 5-6
interfaces as possible oligomerisationsites. This
information might be used to develop novel TGR5 ligands with
a reduced side-effectprofile.},
month = {Feb},
date = {2018-02-22},
organization = {NIC Symposium 2018, Jülich (Germany),
22 Feb 2018 - 23 Feb 2018},
cin = {JSC / NIC / ICS-6},
cid = {I:(DE-Juel1)JSC-20090406 / I:(DE-Juel1)NIC-20090406 /
I:(DE-Juel1)ICS-6-20110106},
pnm = {511 - Computational Science and Mathematical Methods
(POF3-511) / 553 - Physical Basis of Diseases (POF3-553) /
574 - Theory, modelling and simulation (POF3-574) /
Antagonists of the TGR5 G-protein complex formation
$(hdd15_20170501)$ / Energetics of the dimerization and
G-protein coupling of the bile-acid sensing GPCR TGR5
$(hdd15_20160501)$},
pid = {G:(DE-HGF)POF3-511 / G:(DE-HGF)POF3-553 /
G:(DE-HGF)POF3-574 / $G:(DE-Juel1)hdd15_20170501$ /
$G:(DE-Juel1)hdd15_20160501$},
typ = {PUB:(DE-HGF)8 / PUB:(DE-HGF)7},
url = {https://juser.fz-juelich.de/record/844071},
}