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@TECHREPORT{Schmid:845599,
author = {Schmid, U.},
title = {{X}ylosevergärung mit dem thermophilen {B}akterium
{T}hermoanaerobacter {F}innii},
volume = {2168},
number = {Juel-2168},
address = {Jülich},
publisher = {Kernforschungsanlage Jülich, Verlag},
reportid = {FZJ-2018-02819, Juel-2168},
series = {Berichte der Kernforschungsanlage Jülich},
pages = {129 p.},
year = {1987},
abstract = {This study presents results of the fermentation of xylose
with the thermophilic bacterium $\textit{Thermoanaerobacter
finnii}$, especially the effects of substrate concentration
on end product formation. 1. Comparative studies on
differences in morphological and physiological features,
GC-content and DNA-homologies of $\textit{AKO-1}$ with other
thermophilic bacteria were performed. The newly isolated
bacterium $\textit{AKO-1}$ is closely related to the
nonsporeformer $\textit{Thermoanaerobacter ethanolicus}$. It
was classified as $\textit{Thermoanaerobacter finnii}$. 2.
Fermentation of xylose yields ethanol, acetate, L-lactate,
H$_{2}$ and CO$_{2}$ in varying amounts depending on the
substrate concentration. Batch fermentations with increasing
xylose concentrations higher than 66 mM resulted in a
decrease of the ethanol yield from 1.25 to 0.85 mol/mol
xylose (maximum ethanol yield is 1.66 mol/mol xylose) and in
an increase in lactate formation. Fed-batch-fermentations
with stationary concentrations of xylose between 20 and 60
mM lead to a very low lactate production (2mM), so that from
200 mM of xylose 230 mM ethanol were produced, corresponding
to 70\% of the maximal achievable yield. Increasing xylose
consumption rates lead to increasing lactate formation rates
(from 0.04 to 1.52 mmol/ 1-h), whereas the ethanol formation
rate remained largely constant (5.2 and 4.6 mmol/ 1-h
respectively). The inhibitory effect of ethanol on growth
and product formation was circumvented by continous removal
of the produced alcohol from the culture broth by an inert
gas stream. That way xylose consumption could be raised from
200 to 400 mM. 3. Enzymatic studies showed that $\textit{T.
finnii}$ catabolises xylose to xylulose and
xylulose-5-phosphate which is then degraded to pyruvate via
the pentosephosphate pathway and glycolysis.
Fructose-1,6-bisphosphate has been shown to be an activator
of the lactate-dehydrogenase and causes a decrease of the
K$_{M}$ values for NADH and pyruvate. The
lactate-dehydrogenase has a pH-optimum at pH7.0 and is
completely inhibited by 2 mM NADPH. The activity of the
ferredoxin-NAD-reductase (1.1 U/mg protein) was 10 times
higher than the ferredoxin-NADP-reductase activity. For the
first time a transhydrogenase activity (0,9 U/mg protein)
was detected in thermophilic ethanol producing bacteria,
which transfered electrons from NADH to NADP. The activity
of the acetaldehyde-dehydrogenase in $\textit{T. finnii}$
was very low (0.03 U/mg protein) compared to the other
catabolic enzyme activities. 4. The intracellular pyridine
nucleotide levels were between 1.5 to 4 nmol NADPH/mg dry
weight and 0.6 to 2 nmol NADH/mg dry weight, whereas the NAD
and NADP levels reached only 0.1 to 1.2 and 0.5 to 1.2
nmol/mg dry weight, respectively. The intracellular
fructose-1,6-bisphosphate concentrations ranged between 1.2
and 8.4 nmol/mg dry weight. These fructose-1,6-bisphosphate
levels are sufficient for complete activation of the
lactate-dehydrogenase, so that presumably the
lactateformation depends $\underline{not}$
$\underline{only}$ on the intracellular
fructose-1,6-bisphosphate level, but also on the
intracellular concentration of pyruvate.},
cin = {PRE-2000},
cid = {I:(DE-Juel1)PRE2000-20140101},
pnm = {899 - ohne Topic (POF3-899)},
pid = {G:(DE-HGF)POF3-899},
typ = {PUB:(DE-HGF)3 / PUB:(DE-HGF)29},
url = {https://juser.fz-juelich.de/record/845599},
}